pubmed-article:18698139 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18698139 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:18698139 | lifeskim:mentions | umls-concept:C0018801 | lld:lifeskim |
pubmed-article:18698139 | lifeskim:mentions | umls-concept:C0031678 | lld:lifeskim |
pubmed-article:18698139 | lifeskim:mentions | umls-concept:C0683325 | lld:lifeskim |
pubmed-article:18698139 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
pubmed-article:18698139 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
pubmed-article:18698139 | lifeskim:mentions | umls-concept:C0205349 | lld:lifeskim |
pubmed-article:18698139 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:18698139 | pubmed:dateCreated | 2009-2-2 | lld:pubmed |
pubmed-article:18698139 | pubmed:abstractText | A human protein phosphatase inhibitor-1 polymorphism, G147D (c.440G>A, p.147G>D), has been previously demonstrated to blunt the contractile responses of cardiomyocytes to beta-adrenergic agonists. The present study sought to examine whether the G147D inhibitor-1 polymorphism may be associated with specific clinical characteristics of heart failure carriers. | lld:pubmed |
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pubmed-article:18698139 | pubmed:language | eng | lld:pubmed |
pubmed-article:18698139 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18698139 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:18698139 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18698139 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18698139 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18698139 | pubmed:issn | 1421-9751 | lld:pubmed |
pubmed-article:18698139 | pubmed:author | pubmed-author:KraniasEvange... | lld:pubmed |
pubmed-article:18698139 | pubmed:author | pubmed-author:ChenGuoliG | lld:pubmed |
pubmed-article:18698139 | pubmed:author | pubmed-author:PathakAnandA | lld:pubmed |
pubmed-article:18698139 | pubmed:author | pubmed-author:DornGerald... | lld:pubmed |
pubmed-article:18698139 | pubmed:author | pubmed-author:ZhouXiaoyangX | lld:pubmed |
pubmed-article:18698139 | pubmed:copyrightInfo | Copyright 2008 S. Karger AG, Basel. | lld:pubmed |
pubmed-article:18698139 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:18698139 | pubmed:volume | 112 | lld:pubmed |
pubmed-article:18698139 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18698139 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18698139 | pubmed:pagination | 224-31 | lld:pubmed |
pubmed-article:18698139 | pubmed:dateRevised | 2010-9-21 | lld:pubmed |
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pubmed-article:18698139 | pubmed:meshHeading | pubmed-meshheading:18698139... | lld:pubmed |
pubmed-article:18698139 | pubmed:year | 2009 | lld:pubmed |
pubmed-article:18698139 | pubmed:articleTitle | The human G147D-protein phosphatase 1 inhibitor-1 polymorphism is not associated with altered clinical characteristics in heart failure. | lld:pubmed |
pubmed-article:18698139 | pubmed:affiliation | Department of Pharmacology and Cell Biophysics, University of Cincinnati College of Medicine, Cincinnati, Ohio 45267-0575, USA. | lld:pubmed |
pubmed-article:18698139 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:18698139 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:18698139 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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