The aim of this study was to compare the ability of intracoronary adenosine (ADE) and postocclusion hyperemia (PH) to cause maximal hyperemia in humans.
Service de Physiologie et d'Explorations Fonctionnelles, CHU Jean Verdier, Assistance Publique-Hôpitaux de Paris, Université Paris 13, Bondy, France. alain.nitenberg@jvr.aphp.fr
