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| pubmed-article:16845637 | lifeskim:mentions | umls-concept:C0012655 | lld:lifeskim |
| pubmed-article:16845637 | lifeskim:mentions | umls-concept:C0021760 | lld:lifeskim |
| pubmed-article:16845637 | lifeskim:mentions | umls-concept:C0026724 | lld:lifeskim |
| pubmed-article:16845637 | lifeskim:mentions | umls-concept:C0023283 | lld:lifeskim |
| pubmed-article:16845637 | lifeskim:mentions | umls-concept:C0006137 | lld:lifeskim |
| pubmed-article:16845637 | lifeskim:mentions | umls-concept:C0086860 | lld:lifeskim |
| pubmed-article:16845637 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:16845637 | lifeskim:mentions | umls-concept:C0392752 | lld:lifeskim |
| pubmed-article:16845637 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:16845637 | pubmed:dateCreated | 2006-7-17 | lld:pubmed |
| pubmed-article:16845637 | pubmed:abstractText | Mucosal leishmaniasis (ML) is associated with exaggerated tumor necrosis factor- alpha and interferon- gamma responses and tissue destruction. ML follows localized cutaneous leishmaniasis (CL) caused by Leishmania braziliensis infection. Interleukin (IL)-6 down-regulates T helper (Th) cell type 1 differentiation and drives Th2 cell differentiation. The IL6 -174 G/C polymorphism is associated with proinflammatory diseases and IL-6 regulation. | lld:pubmed |
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| pubmed-article:16845637 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16845637 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16845637 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:16845637 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16845637 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:16845637 | pubmed:issn | 0022-1899 | lld:pubmed |
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| pubmed-article:16845637 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16845637 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:16845637 | pubmed:volume | 194 | lld:pubmed |
| pubmed-article:16845637 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16845637 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16845637 | pubmed:pagination | 519-27 | lld:pubmed |
| pubmed-article:16845637 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:16845637 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16845637 | pubmed:articleTitle | IL6 -174 G/C promoter polymorphism influences susceptibility to mucosal but not localized cutaneous leishmaniasis in Brazil. | lld:pubmed |
| pubmed-article:16845637 | pubmed:affiliation | Servico de Imunologia, Hospital Universitario Professor Edgard Santos, Universidade Federal da Bahia, Salvador, BA, Brazil. | lld:pubmed |
| pubmed-article:16845637 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16845637 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:16845637 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:16845637 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| pubmed-article:16845637 | pubmed:publicationType | Research Support, N.I.H., Intramural | lld:pubmed |
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