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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
2006-2-28
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pubmed:abstractText |
Complement has been implicated in the pathogenesis of neurodegenerative diseases. The purpose of this study was to investigate whether complement activation is part of the pathogenesis of retinal ganglion cell (RGC) loss in glaucoma.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
0146-0404
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
47
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1024-9
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:16505037-Animals,
pubmed-meshheading:16505037-Cell Death,
pubmed-meshheading:16505037-Complement C1q,
pubmed-meshheading:16505037-Disease Models, Animal,
pubmed-meshheading:16505037-Female,
pubmed-meshheading:16505037-Fluorescent Antibody Technique, Indirect,
pubmed-meshheading:16505037-Glaucoma,
pubmed-meshheading:16505037-Humans,
pubmed-meshheading:16505037-Immunoblotting,
pubmed-meshheading:16505037-Macaca fascicularis,
pubmed-meshheading:16505037-Male,
pubmed-meshheading:16505037-Mice,
pubmed-meshheading:16505037-Mice, Inbred C57BL,
pubmed-meshheading:16505037-Mice, Inbred DBA,
pubmed-meshheading:16505037-Middle Aged,
pubmed-meshheading:16505037-RNA, Messenger,
pubmed-meshheading:16505037-Retina,
pubmed-meshheading:16505037-Retinal Ganglion Cells,
pubmed-meshheading:16505037-Reverse Transcriptase Polymerase Chain Reaction,
pubmed-meshheading:16505037-Up-Regulation
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pubmed:year |
2006
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pubmed:articleTitle |
Complement component 1Q (C1Q) upregulation in retina of murine, primate, and human glaucomatous eyes.
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pubmed:affiliation |
Department of Ophthalmology, Mt. Sinai School of Medicine, New York, New York 10029, USA.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
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