pubmed-article:10704237 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:10704237 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
pubmed-article:10704237 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
pubmed-article:10704237 | lifeskim:mentions | umls-concept:C0017638 | lld:lifeskim |
pubmed-article:10704237 | lifeskim:mentions | umls-concept:C0021641 | lld:lifeskim |
pubmed-article:10704237 | lifeskim:mentions | umls-concept:C0253023 | lld:lifeskim |
pubmed-article:10704237 | lifeskim:mentions | umls-concept:C0376515 | lld:lifeskim |
pubmed-article:10704237 | lifeskim:mentions | umls-concept:C1539477 | lld:lifeskim |
pubmed-article:10704237 | lifeskim:mentions | umls-concept:C1515370 | lld:lifeskim |
pubmed-article:10704237 | lifeskim:mentions | umls-concept:C1314939 | lld:lifeskim |
pubmed-article:10704237 | lifeskim:mentions | umls-concept:C0205374 | lld:lifeskim |
pubmed-article:10704237 | pubmed:issue | 8 | lld:pubmed |
pubmed-article:10704237 | pubmed:dateCreated | 2000-4-11 | lld:pubmed |
pubmed-article:10704237 | pubmed:abstractText | The expression of fas gene in glioma cells varies with growth stage. When insulin-elicited transient apoptosis of glioma cells was in progress, the expression of fas gene increased at both transcriptional and translational levels. In contrast, the expression of fas-L gene in glioma cells remained constant. Apoptosis occurred in the cells having high level of surface Fas protein. When the expression of Fas-L in U-373MG cells was suppressed by ribozyme, the insulin-elicited transient apoptosis vanished. Overexpression of Bcl-2 in U-373MG cells did not alter significantly the cell cycle progression and the expression of fas gene. However, these cells were resistant to insulin-trigged death. Therefore, insulin-elicited apoptosis involved Fas-related death signal, and which could be prevented by the protective effect of Bcl-2. | lld:pubmed |
pubmed-article:10704237 | pubmed:language | eng | lld:pubmed |
pubmed-article:10704237 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10704237 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:10704237 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10704237 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10704237 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10704237 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10704237 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10704237 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10704237 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10704237 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10704237 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10704237 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10704237 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:10704237 | pubmed:issn | 1065-6995 | lld:pubmed |
pubmed-article:10704237 | pubmed:author | pubmed-author:WangC HCH | lld:pubmed |
pubmed-article:10704237 | pubmed:author | pubmed-author:WangY SYS | lld:pubmed |
pubmed-article:10704237 | pubmed:author | pubmed-author:HsüM CMC | lld:pubmed |
pubmed-article:10704237 | pubmed:author | pubmed-author:YangB CBC | lld:pubmed |
pubmed-article:10704237 | pubmed:author | pubmed-author:TangM JMJ | lld:pubmed |
pubmed-article:10704237 | pubmed:author | pubmed-author:YangT LTL | lld:pubmed |
pubmed-article:10704237 | pubmed:copyrightInfo | Copyright 1999 Academic Press. | lld:pubmed |
pubmed-article:10704237 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:10704237 | pubmed:volume | 23 | lld:pubmed |
pubmed-article:10704237 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:10704237 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:10704237 | pubmed:pagination | 533-40 | lld:pubmed |
pubmed-article:10704237 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:10704237 | pubmed:year | 1999 | lld:pubmed |
pubmed-article:10704237 | pubmed:articleTitle | Transient apoptosis elicited by insulin in serum-starved glioma cells involves Fas/Fas-L and Bcl-2. | lld:pubmed |
pubmed-article:10704237 | pubmed:affiliation | Department of Microbiology and Immunology, College of Medicine, National Cheng Kung University, Tainan, Taiwan, Republic of China. y1357@mail.ncku.edu.tw | lld:pubmed |
pubmed-article:10704237 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:10704237 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10704237 | lld:pubmed |