| pubmed-article:10430757 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10430757 | lifeskim:mentions | umls-concept:C1273518 | lld:lifeskim |
| pubmed-article:10430757 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:10430757 | lifeskim:mentions | umls-concept:C0011696 | lld:lifeskim |
| pubmed-article:10430757 | lifeskim:mentions | umls-concept:C0264793 | lld:lifeskim |
| pubmed-article:10430757 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:10430757 | pubmed:dateCreated | 1999-8-13 | lld:pubmed |
| pubmed-article:10430757 | pubmed:abstractText | Idiopathic dilated cardiomyopathy, of which approximately 20% of cases are familial (FDCM), is a primary myocardial disorder characterized by ventricular dilatation and impaired systolic function. It is a common cause of heart failure and the need for cardiac transplantation. Although 6 chromosomal loci responsible for autosomal dominant FDCM have been mapped by linkage analysis, none of these genes have been identified. By use of the candidate-gene approach, actin was identified recently as being responsible for dilated cardiomyopathy. Considerable evidence suggests desmin, a muscle-specific intermediate filament, plays a significant role in cardiac growth and development. | lld:pubmed |
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| pubmed-article:10430757 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10430757 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10430757 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10430757 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:10430757 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10430757 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:10430757 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:10430757 | pubmed:author | pubmed-author:HillRR | lld:pubmed |
| pubmed-article:10430757 | pubmed:author | pubmed-author:RobertiDD | lld:pubmed |
| pubmed-article:10430757 | pubmed:author | pubmed-author:PILZCC | lld:pubmed |
| pubmed-article:10430757 | pubmed:author | pubmed-author:GonzalesVV | lld:pubmed |
| pubmed-article:10430757 | pubmed:author | pubmed-author:LUPP | lld:pubmed |
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| pubmed-article:10430757 | pubmed:author | pubmed-author:ZoghbiW AWA | lld:pubmed |
| pubmed-article:10430757 | pubmed:author | pubmed-author:QuiñonesM AMA | lld:pubmed |
| pubmed-article:10430757 | pubmed:author | pubmed-author:BurchP EPE | lld:pubmed |
| pubmed-article:10430757 | pubmed:author | pubmed-author:TapscoftTT | lld:pubmed |
| pubmed-article:10430757 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:10430757 | pubmed:day | 3 | lld:pubmed |
| pubmed-article:10430757 | pubmed:volume | 100 | lld:pubmed |
| pubmed-article:10430757 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10430757 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10430757 | pubmed:pagination | 461-4 | lld:pubmed |
| pubmed-article:10430757 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:10430757 | pubmed:meshHeading | pubmed-meshheading:10430757... | lld:pubmed |
| pubmed-article:10430757 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10430757 | pubmed:articleTitle | Desmin mutation responsible for idiopathic dilated cardiomyopathy. | lld:pubmed |
| pubmed-article:10430757 | pubmed:affiliation | Section of Cardiology, Molecular Biology Computational Resource, Baylor College of Medicine, Houston, TX, USA. | lld:pubmed |
| pubmed-article:10430757 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10430757 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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