pubmed-article:9889795 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:9889795 | lifeskim:mentions | umls-concept:C0014257 | lld:lifeskim |
pubmed-article:9889795 | lifeskim:mentions | umls-concept:C0273115 | lld:lifeskim |
pubmed-article:9889795 | lifeskim:mentions | umls-concept:C0003765 | lld:lifeskim |
pubmed-article:9889795 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
pubmed-article:9889795 | lifeskim:mentions | umls-concept:C0599946 | lld:lifeskim |
pubmed-article:9889795 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:9889795 | pubmed:dateCreated | 1999-1-28 | lld:pubmed |
pubmed-article:9889795 | pubmed:abstractText | Pulmonary vasorelaxation to endothelium-dependent and independent agonists is dysfunctional in endotoxin-induced acute lung injury. L-arginine is the precursor to endothelial production of nitric oxide (NO), suggesting that arginine and NO are intimately linked. We hypothesized that L-arginine would attenuate endotoxin-induced dysfunction of guanosine 3',5'-cyclic monophosphate-mediated pulmonary vasorelaxation. | lld:pubmed |
pubmed-article:9889795 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9889795 | pubmed:language | eng | lld:pubmed |
pubmed-article:9889795 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9889795 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:9889795 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9889795 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9889795 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9889795 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9889795 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9889795 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9889795 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:9889795 | pubmed:month | Jan | lld:pubmed |
pubmed-article:9889795 | pubmed:issn | 0039-6060 | lld:pubmed |
pubmed-article:9889795 | pubmed:author | pubmed-author:MeldrumD RDR | lld:pubmed |
pubmed-article:9889795 | pubmed:author | pubmed-author:FullertonD... | lld:pubmed |
pubmed-article:9889795 | pubmed:author | pubmed-author:McIntyreR... | lld:pubmed |
pubmed-article:9889795 | pubmed:author | pubmed-author:SheridanB CBC | lld:pubmed |
pubmed-article:9889795 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:9889795 | pubmed:volume | 125 | lld:pubmed |
pubmed-article:9889795 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:9889795 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:9889795 | pubmed:pagination | 33-40 | lld:pubmed |
pubmed-article:9889795 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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pubmed-article:9889795 | pubmed:year | 1999 | lld:pubmed |
pubmed-article:9889795 | pubmed:articleTitle | L-arginine attenuates endothelial dysfunction in endotoxin-induced lung injury. | lld:pubmed |
pubmed-article:9889795 | pubmed:affiliation | Department of Surgery, University of Colorado, Denver, USA. | lld:pubmed |
pubmed-article:9889795 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:9889795 | pubmed:publicationType | In Vitro | lld:pubmed |
pubmed-article:9889795 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:9889795 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |