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pubmed-article:9889795pubmed:abstractTextPulmonary vasorelaxation to endothelium-dependent and independent agonists is dysfunctional in endotoxin-induced acute lung injury. L-arginine is the precursor to endothelial production of nitric oxide (NO), suggesting that arginine and NO are intimately linked. We hypothesized that L-arginine would attenuate endotoxin-induced dysfunction of guanosine 3',5'-cyclic monophosphate-mediated pulmonary vasorelaxation.lld:pubmed
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pubmed-article:9889795pubmed:articleTitleL-arginine attenuates endothelial dysfunction in endotoxin-induced lung injury.lld:pubmed
pubmed-article:9889795pubmed:affiliationDepartment of Surgery, University of Colorado, Denver, USA.lld:pubmed
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