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pubmed-article:959508rdf:typepubmed:Citationlld:pubmed
pubmed-article:959508lifeskim:mentionsumls-concept:C0341628lld:lifeskim
pubmed-article:959508lifeskim:mentionsumls-concept:C0002978lld:lifeskim
pubmed-article:959508pubmed:issue6lld:pubmed
pubmed-article:959508pubmed:dateCreated1976-10-20lld:pubmed
pubmed-article:959508pubmed:abstractText119 kidneys demonstrating changes consistent with renal tuberculosis were studied in a total of 94 patients by means of selective, transfemoral renal antiography. In the first stage of the disease, this method detected parenchymal involvement which could not be visualized by excretory urography. In stage II the arcuate arteries and occasionally the intralobular arteries, showed typical changes. Cavitation, pathognomonic for the presence of renal tuberculosis were found in 39.3% of these cases. In 40% of the cases the angiographic findingd were more extensive than the apparent findings of excretory urography. Angiography, thus, can render valuable information pertaining to the course of the disease and the necessity of operative intervention. Stage III was usually characterized by extensive changes specific for parenchymal destruction. Typical vascular lesions were readily recognized. Cavitation was found in 86.5% of these cases. Tortuosity and dilatation of the renal pelvis- and/or ureter-arteries revealed evidence of ureteral involvement (stage III2). In the presence of a non-functioning kidney angiography is mandatory to rule out renal aplasia and to differentiate between a kidney destroyed by other disease processes and the complete cavitary destruction of end-stage renal tuberculosis (stage III3).lld:pubmed
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pubmed-article:959508pubmed:journalhttp://linkedlifedata.com/r...lld:pubmed
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pubmed-article:959508pubmed:statusMEDLINElld:pubmed
pubmed-article:959508pubmed:monthJunlld:pubmed
pubmed-article:959508pubmed:issn0033-832Xlld:pubmed
pubmed-article:959508pubmed:authorpubmed-author:KleinUUlld:pubmed
pubmed-article:959508pubmed:authorpubmed-author:LissnerJJlld:pubmed
pubmed-article:959508pubmed:authorpubmed-author:EisenbergerFFlld:pubmed
pubmed-article:959508pubmed:authorpubmed-author:HeinzeH GHGlld:pubmed
pubmed-article:959508pubmed:authorpubmed-author:PfeiferK JKJlld:pubmed
pubmed-article:959508pubmed:authorpubmed-author:RunteRRlld:pubmed
pubmed-article:959508pubmed:authorpubmed-author:ThymWWlld:pubmed
pubmed-article:959508pubmed:issnTypePrintlld:pubmed
pubmed-article:959508pubmed:volume16lld:pubmed
pubmed-article:959508pubmed:ownerNLMlld:pubmed
pubmed-article:959508pubmed:authorsCompleteYlld:pubmed
pubmed-article:959508pubmed:pagination240-7lld:pubmed
pubmed-article:959508pubmed:dateRevised2007-11-15lld:pubmed
pubmed-article:959508pubmed:meshHeadingpubmed-meshheading:959508-H...lld:pubmed
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pubmed-article:959508pubmed:meshHeadingpubmed-meshheading:959508-A...lld:pubmed
pubmed-article:959508pubmed:meshHeadingpubmed-meshheading:959508-R...lld:pubmed
pubmed-article:959508pubmed:meshHeadingpubmed-meshheading:959508-T...lld:pubmed
pubmed-article:959508pubmed:year1976lld:pubmed
pubmed-article:959508pubmed:articleTitle[Angiography of urotuberculosis (author's transl)].lld:pubmed
pubmed-article:959508pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:959508pubmed:publicationTypeEnglish Abstractlld:pubmed