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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:issue |
3
|
| pubmed:dateCreated |
1997-3-11
|
| pubmed:abstractText |
Disease-associated electrophysiological alterations may contribute to the increased predisposition to arrhythmias of the hypertrophied or failing myocardium. An I(f)-like current is expressed in rat left ventricular myocytes (LVMs), its amplitude being linearly related to the severity of cardiac hypertrophy. Here, we report the occurrence and electrophysiological properties of I(f) in human LVMs.
|
| pubmed:language |
eng
|
| pubmed:journal | |
| pubmed:citationSubset |
AIM
|
| pubmed:chemical | |
| pubmed:status |
MEDLINE
|
| pubmed:month |
Feb
|
| pubmed:issn |
0009-7322
|
| pubmed:author | |
| pubmed:issnType |
Print
|
| pubmed:day |
4
|
| pubmed:volume |
95
|
| pubmed:owner |
NLM
|
| pubmed:authorsComplete |
Y
|
| pubmed:pagination |
568-71
|
| pubmed:dateRevised |
2006-11-15
|
| pubmed:meshHeading |
pubmed-meshheading:9024140-Cardiac Output, Low,
pubmed-meshheading:9024140-Cesium,
pubmed-meshheading:9024140-Electrophysiology,
pubmed-meshheading:9024140-Extracellular Space,
pubmed-meshheading:9024140-Humans,
pubmed-meshheading:9024140-Male,
pubmed-meshheading:9024140-Middle Aged,
pubmed-meshheading:9024140-Myocardium,
pubmed-meshheading:9024140-Potassium,
pubmed-meshheading:9024140-Ventricular Function, Left
|
| pubmed:year |
1997
|
| pubmed:articleTitle |
Characterization of the hyperpolarization-activated current, I(f), in ventricular myocytes from human failing heart.
|
| pubmed:affiliation |
Department of Pharmacology, University of Firenze, Italy.
|
| pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|