rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
2
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pubmed:dateCreated |
1993-8-30
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pubmed:abstractText |
Hyperkalemic depolarized cardiac arrest has been the cornerstone of myocardial protection during cardiac surgery for more than 30 years. Many of the advances in myocardial protection seek to minimize the cellular damage and to reduce the ongoing metabolic processes occurring as a direct consequence of the depolarized state. Ideally, cardiac arrest at hyperpolarized cellular membrane potentials--the natural resting state of the heart--will meet all the requirements of modern cardioplegia, namely, electromechanical asystole and cardiac relaxation, while preserving the vital integrity of the heart itself.
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pubmed:grant |
|
pubmed:commentsCorrections |
|
pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
AIM
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pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Aug
|
pubmed:issn |
0022-5223
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pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
106
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
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pubmed:pagination |
317-28
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:7848379-Adenosine Triphosphate,
pubmed-meshheading:7848379-Animals,
pubmed-meshheading:7848379-Dose-Response Relationship, Drug,
pubmed-meshheading:7848379-Female,
pubmed-meshheading:7848379-Heart Arrest,
pubmed-meshheading:7848379-Male,
pubmed-meshheading:7848379-Models, Cardiovascular,
pubmed-meshheading:7848379-Myocardial Ischemia,
pubmed-meshheading:7848379-Picolines,
pubmed-meshheading:7848379-Potassium Channels,
pubmed-meshheading:7848379-Pyrans,
pubmed-meshheading:7848379-Rabbits
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pubmed:year |
1993
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pubmed:articleTitle |
Elective cardiac arrest with a hyperpolarizing adenosine triphosphate-sensitive potassium channel opener. A novel form of myocardial protection?
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pubmed:affiliation |
Department of Surgery, Medical College of Virginia, Richmond 23298-0645.
|
pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
|