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| pubmed-article:6264122 | lifeskim:mentions | umls-concept:C0000742 | lld:lifeskim |
| pubmed-article:6264122 | lifeskim:mentions | umls-concept:C0033755 | lld:lifeskim |
| pubmed-article:6264122 | lifeskim:mentions | umls-concept:C0016030 | lld:lifeskim |
| pubmed-article:6264122 | lifeskim:mentions | umls-concept:C0086574 | lld:lifeskim |
| pubmed-article:6264122 | lifeskim:mentions | umls-concept:C0017428 | lld:lifeskim |
| pubmed-article:6264122 | lifeskim:mentions | umls-concept:C0205419 | lld:lifeskim |
| pubmed-article:6264122 | lifeskim:mentions | umls-concept:C0678594 | lld:lifeskim |
| pubmed-article:6264122 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:6264122 | pubmed:dateCreated | 1981-8-10 | lld:pubmed |
| pubmed-article:6264122 | pubmed:abstractText | We have prepared full-length DNA clones of the Abelson murine leukemia virus (A-MuLV) genome. A specific probe homologous to the central portion of the A-MuLV genome was prepared by nick translation of a subcloned restriction fraction from the cloned DNA. The probe was used to examine the genome structure of several A-MuLV variants. The conclusions are: (i) three viruses coding for Abelson-specific proteins of molecular weight 120,000, 100,000, and 90,000 had genomes indistinguishable in size, suggesting that the shorter proteins are the result of early translational termination; (ii) compared with the genome encoding the 120,000-dalton (120K) protein, a genome coding for a 160K protein was 0.8 kilobase larger in the A-MuLV-specific region; and (iii) a genome coding for a 92K protein had a 700-base pair deletion internal to the coding region. This mutant was transformation defective: its 92K protein lacked the protein kinase activity normally associated with the A-MuLV protein, and cells containing the virus were not morphologically transformed. In addition, we determined the number of A-MuLV proviruses in each of several transformed fibroblast and lymphoid cells prepared by infection in vitro. These experiments show that a single copy of the A-MuLV provirus is sufficient to transform both types of cells and that nonproducer cells generally have only one integrated provirus. | lld:pubmed |
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| pubmed-article:6264122 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:6264122 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:6264122 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:6264122 | pubmed:month | May | lld:pubmed |
| pubmed-article:6264122 | pubmed:issn | 0022-538X | lld:pubmed |
| pubmed-article:6264122 | pubmed:author | pubmed-author:BaltimoreDD | lld:pubmed |
| pubmed-article:6264122 | pubmed:author | pubmed-author:WitteO NON | lld:pubmed |
| pubmed-article:6264122 | pubmed:author | pubmed-author:GilboaEE | lld:pubmed |
| pubmed-article:6264122 | pubmed:author | pubmed-author:RosenbergNN | lld:pubmed |
| pubmed-article:6264122 | pubmed:author | pubmed-author:GoesS JSJ | lld:pubmed |
| pubmed-article:6264122 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:6264122 | pubmed:volume | 38 | lld:pubmed |
| pubmed-article:6264122 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:6264122 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:6264122 | pubmed:pagination | 460-8 | lld:pubmed |
| pubmed-article:6264122 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:6264122 | pubmed:year | 1981 | lld:pubmed |
| pubmed-article:6264122 | pubmed:articleTitle | Genome structure of Abelson murine leukemia virus variants: proviruses in fibroblasts and lymphoid cells. | lld:pubmed |
| pubmed-article:6264122 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:6264122 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:6264122 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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