| pubmed-article:3865197 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3865197 | lifeskim:mentions | umls-concept:C0007090 | lld:lifeskim |
| pubmed-article:3865197 | lifeskim:mentions | umls-concept:C1522424 | lld:lifeskim |
| pubmed-article:3865197 | lifeskim:mentions | umls-concept:C1336745 | lld:lifeskim |
| pubmed-article:3865197 | lifeskim:mentions | umls-concept:C0205307 | lld:lifeskim |
| pubmed-article:3865197 | lifeskim:mentions | umls-concept:C0002085 | lld:lifeskim |
| pubmed-article:3865197 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:3865197 | lifeskim:mentions | umls-concept:C1517945 | lld:lifeskim |
| pubmed-article:3865197 | pubmed:issue | 23 | lld:pubmed |
| pubmed-article:3865197 | pubmed:dateCreated | 1986-1-6 | lld:pubmed |
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| pubmed-article:3865197 | pubmed:abstractText | Young mice injected with the carcinogen N-nitroso-N-methylurea develop thymic lymphomas 2-4 months later. We previously have shown that these tumors frequently contain an activated N-ras gene that can transform rodent fibroblasts in vitro. We report here the intron/exon structure of such an activated N-ras gene and the sequence of its four coding exons. A single nucleotide change is responsible for the transforming alteration, a C----A transversion in the first base of codon 61. Through the use of synthetic oligonucleotides as hybridization probes, we show that this tumor lacks the normal allele of the N-ras gene. The implications of this finding for oncogene dominance are discussed. | lld:pubmed |
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| pubmed-article:3865197 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3865197 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3865197 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:3865197 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3865197 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3865197 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:3865197 | pubmed:issn | 0027-8424 | lld:pubmed |
| pubmed-article:3865197 | pubmed:author | pubmed-author:PellicerAA | lld:pubmed |
| pubmed-article:3865197 | pubmed:author | pubmed-author:VillasanteAA | lld:pubmed |
| pubmed-article:3865197 | pubmed:author | pubmed-author:GuerreroII | lld:pubmed |
| pubmed-article:3865197 | pubmed:author | pubmed-author:CorcesVV | lld:pubmed |
| pubmed-article:3865197 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3865197 | pubmed:volume | 82 | lld:pubmed |
| pubmed-article:3865197 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3865197 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3865197 | pubmed:pagination | 7810-4 | lld:pubmed |
| pubmed-article:3865197 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:3865197 | pubmed:meshHeading | pubmed-meshheading:3865197-... | lld:pubmed |
| pubmed-article:3865197 | pubmed:year | 1985 | lld:pubmed |
| pubmed-article:3865197 | pubmed:articleTitle | Loss of the normal N-ras allele in a mouse thymic lymphoma induced by a chemical carcinogen. | lld:pubmed |
| pubmed-article:3865197 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3865197 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:3865197 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:3865197 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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