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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
2
|
pubmed:dateCreated |
1979-12-20
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pubmed:abstractText |
Bloodstream forms of Trypanosoma cruzi had a substantial increase in respiration in the presence of acetate. Oxidation of acetate took place via the tricarboxylic acid cycle and involved an antimycin A-sensitive respiratory pathway. Oxygen uptake in the presence of acetate was a sensitive to antimycin A inhibition as was CO2 production. There was a 6--7% residual O2 uptake which was not inhibited by high antimycin concentrations. Human anti-T. cruzi sera had no effect on oxygen uptake.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
0022-3921
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
26
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
301-3
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pubmed:dateRevised |
2003-11-14
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pubmed:meshHeading |
pubmed-meshheading:385857-Acetates,
pubmed-meshheading:385857-Animals,
pubmed-meshheading:385857-Antigen-Antibody Reactions,
pubmed-meshheading:385857-Antimycin A,
pubmed-meshheading:385857-Blood,
pubmed-meshheading:385857-Immune Sera,
pubmed-meshheading:385857-Mice,
pubmed-meshheading:385857-Oxidation-Reduction,
pubmed-meshheading:385857-Oxygen Consumption,
pubmed-meshheading:385857-Trypanosoma cruzi
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pubmed:year |
1979
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pubmed:articleTitle |
Acetate oxidation by bloodstream forms of Trypanosoma cruzi.
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pubmed:publicationType |
Journal Article
|