rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
2-3
|
pubmed:dateCreated |
1989-9-11
|
pubmed:abstractText |
Unilateral lesions of the substantia nigra were performed on baboons which had been previously trained to perform a visual pointing movement. The resulting motor impairment consisted of an increase in movement duration. This bradykinesia shows as a regular decrease in velocity. A possible change in mode of motor control caused by lesion of the SN is discussed. Partial exclusion of SN might bring about a shift from the feedforward to a feedback mode relaying more heavily on visual cues.
|
pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:issn |
0006-8977
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
33
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
183-8
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:2758300-Acceleration,
pubmed-meshheading:2758300-Animals,
pubmed-meshheading:2758300-Arousal,
pubmed-meshheading:2758300-Attention,
pubmed-meshheading:2758300-Corpus Striatum,
pubmed-meshheading:2758300-Cues,
pubmed-meshheading:2758300-Dominance, Cerebral,
pubmed-meshheading:2758300-Dopamine,
pubmed-meshheading:2758300-Feedback,
pubmed-meshheading:2758300-Isometric Contraction,
pubmed-meshheading:2758300-Muscles,
pubmed-meshheading:2758300-Neural Pathways,
pubmed-meshheading:2758300-Orientation,
pubmed-meshheading:2758300-Papio,
pubmed-meshheading:2758300-Psychomotor Performance,
pubmed-meshheading:2758300-Reaction Time,
pubmed-meshheading:2758300-Receptors, Dopamine,
pubmed-meshheading:2758300-Substantia Nigra
|
pubmed:year |
1989
|
pubmed:articleTitle |
Experimental bradykinesia in the monkey: speed control impairments after lesion of the substantia nigra.
|
pubmed:affiliation |
Laboratoire de Neurosciences Fonctionnelles, CNRS, Marseille, France.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|