21561957

Source:http://linkedlifedata.com/resource/pubmed/id/21561957

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0014653, umls-concept:C0026769, umls-concept:C0030705, umls-concept:C0035020, umls-concept:C0087111, umls-concept:C0205178, umls-concept:C1280500, umls-concept:C1521761
pubmed:issue	7
pubmed:dateCreated	2011-6-21
pubmed:abstractText	In multiple sclerosis inflammation is primarily injurious to the central nervous system, but its therapeutic suppression might inhibit repair-promoting factors.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9509185
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Brain-Derived Neurotrophic Factor, http://linkedlifedata.com/resource/pubmed/chemical/Ciliary Neurotrophic Factor, http://linkedlifedata.com/resource/pubmed/chemical/Glucocorticoids, http://linkedlifedata.com/resource/pubmed/chemical/Immunologic Factors, http://linkedlifedata.com/resource/pubmed/chemical/Inflammation Mediators, http://linkedlifedata.com/resource/pubmed/chemical/Interferon-beta, http://linkedlifedata.com/resource/pubmed/chemical/Methylprednisolone, http://linkedlifedata.com/resource/pubmed/chemical/NOS2 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Nerve Growth Factors, http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase Type II, http://linkedlifedata.com/resource/pubmed/chemical/Tumor Necrosis Factor-alpha, http://linkedlifedata.com/resource/pubmed/chemical/interferon beta 1a, http://linkedlifedata.com/resource/pubmed/chemical/interferon beta-1b
pubmed:status	MEDLINE
pubmed:month	Jul
pubmed:issn	1477-0970
pubmed:author	pubmed-author:HassingerSarahS, pubmed-author:LindquistJonathan AJA, pubmed-author:LindquistSabineS, pubmed-author:SailerMichaelM
pubmed:issnType	Electronic
pubmed:volume	17
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	851-66
pubmed:dateRevised	2011-10-27
pubmed:meshHeading	pubmed-meshheading:21561957-Adult, pubmed-meshheading:21561957-Brain-Derived Neurotrophic Factor, pubmed-meshheading:21561957-Case-Control Studies, pubmed-meshheading:21561957-Cell Separation, pubmed-meshheading:21561957-Cells, Cultured, pubmed-meshheading:21561957-Ciliary Neurotrophic Factor, pubmed-meshheading:21561957-Drug Therapy, Combination, pubmed-meshheading:21561957-Female, pubmed-meshheading:21561957-Flow Cytometry, pubmed-meshheading:21561957-Glucocorticoids, pubmed-meshheading:21561957-Humans, pubmed-meshheading:21561957-Immunologic Factors, pubmed-meshheading:21561957-Inflammation Mediators, pubmed-meshheading:21561957-Interferon-beta, pubmed-meshheading:21561957-Leukocytes, Mononuclear, pubmed-meshheading:21561957-Male, pubmed-meshheading:21561957-Methylprednisolone, pubmed-meshheading:21561957-Middle Aged, pubmed-meshheading:21561957-Multiple Sclerosis, Chronic Progressive, pubmed-meshheading:21561957-Multiple Sclerosis, Relapsing-Remitting, pubmed-meshheading:21561957-Nerve Growth Factors, pubmed-meshheading:21561957-Nitric Oxide Synthase Type II, pubmed-meshheading:21561957-Treatment Outcome, pubmed-meshheading:21561957-Tumor Necrosis Factor-alpha, pubmed-meshheading:21561957-Young Adult
pubmed:year	2011
pubmed:articleTitle	The balance of pro-inflammatory and trophic factors in multiple sclerosis patients: effects of acute relapse and immunomodulatory treatment.
pubmed:affiliation	Department of Neurology, Hannover Medical School, Hannover, Germany. Lindquist.Sabine@mh-hannover.de
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't