| pubmed-article:21464990 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21464990 | lifeskim:mentions | umls-concept:C0227525 | lld:lifeskim |
| pubmed-article:21464990 | lifeskim:mentions | umls-concept:C0521451 | lld:lifeskim |
| pubmed-article:21464990 | lifeskim:mentions | umls-concept:C1101610 | lld:lifeskim |
| pubmed-article:21464990 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:21464990 | lifeskim:mentions | umls-concept:C0021655 | lld:lifeskim |
| pubmed-article:21464990 | lifeskim:mentions | umls-concept:C1527148 | lld:lifeskim |
| pubmed-article:21464990 | lifeskim:mentions | umls-concept:C0599894 | lld:lifeskim |
| pubmed-article:21464990 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:21464990 | lifeskim:mentions | umls-concept:C1314939 | lld:lifeskim |
| pubmed-article:21464990 | lifeskim:mentions | umls-concept:C0348080 | lld:lifeskim |
| pubmed-article:21464990 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:21464990 | pubmed:dateCreated | 2011-4-5 | lld:pubmed |
| pubmed-article:21464990 | pubmed:abstractText | Mitochondrial dysfunction induces insulin resistance in myocytes via a reduction of insulin receptor substrate-1 (IRS-1) expression. However, the effect of mitochondrial dysfunction on insulin sensitivity is not understood well in hepatocytes. Although research has implicated the translational repression of target genes by endogenous non-coding microRNAs (miRNA) in the pathogenesis of various diseases, the identity and role of the miRNAs that are involved in the development of insulin resistance also remain largely unknown. | lld:pubmed |
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| pubmed-article:21464990 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:21464990 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:21464990 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21464990 | pubmed:issn | 1932-6203 | lld:pubmed |
| pubmed-article:21464990 | pubmed:author | pubmed-author:MaoZ YZY | lld:pubmed |
| pubmed-article:21464990 | pubmed:author | pubmed-author:ZhangJinJ | lld:pubmed |
| pubmed-article:21464990 | pubmed:author | pubmed-author:ParkSeung-Yoo... | lld:pubmed |
| pubmed-article:21464990 | pubmed:author | pubmed-author:MaDuanD | lld:pubmed |
| pubmed-article:21464990 | pubmed:author | pubmed-author:RyuHyun SuHS | lld:pubmed |
| pubmed-article:21464990 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21464990 | pubmed:volume | 6 | lld:pubmed |
| pubmed-article:21464990 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21464990 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21464990 | pubmed:pagination | e17343 | lld:pubmed |
| pubmed-article:21464990 | pubmed:dateRevised | 2011-8-17 | lld:pubmed |
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| pubmed-article:21464990 | pubmed:meshHeading | pubmed-meshheading:21464990... | lld:pubmed |
| pubmed-article:21464990 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21464990 | pubmed:articleTitle | The induction of microRNA targeting IRS-1 is involved in the development of insulin resistance under conditions of mitochondrial dysfunction in hepatocytes. | lld:pubmed |
| pubmed-article:21464990 | pubmed:affiliation | Department of Biochemistry, Dongguk University College of Medicine, Kyungju, Korea. | lld:pubmed |
| pubmed-article:21464990 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:21464990 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
