| pubmed-article:20970138 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20970138 | lifeskim:mentions | umls-concept:C0026473 | lld:lifeskim |
| pubmed-article:20970138 | lifeskim:mentions | umls-concept:C0600210 | lld:lifeskim |
| pubmed-article:20970138 | lifeskim:mentions | umls-concept:C0871161 | lld:lifeskim |
| pubmed-article:20970138 | lifeskim:mentions | umls-concept:C0050156 | lld:lifeskim |
| pubmed-article:20970138 | lifeskim:mentions | umls-concept:C1998811 | lld:lifeskim |
| pubmed-article:20970138 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:20970138 | pubmed:dateCreated | 2010-12-6 | lld:pubmed |
| pubmed-article:20970138 | pubmed:abstractText | We have previously shown that CLA induces regression of pre-established atherosclerotic lesions in apoE(-/-) mice. CLA is a known ligand of peroxisome proliferator activated receptors (PPARs) and it is postulated that CLA mediates its atheroprotective effects through activation of PPARs. Earlier work in our group identified the monocyte/macrophage cell as the primary cellular target of CLA. In this study we identified novel genes regulated by CLA during the regression of atherosclerosis and characterised a role for one of these, SorLA. SorLA is a member of the vacuolar protein sorting 10 protein (Vps10p) domain receptor family, which has structural homology with the LDLR family. | lld:pubmed |
| pubmed-article:20970138 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20970138 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20970138 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20970138 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:20970138 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20970138 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:20970138 | pubmed:issn | 1879-1484 | lld:pubmed |
| pubmed-article:20970138 | pubmed:author | pubmed-author:FitzgeraldDes... | lld:pubmed |
| pubmed-article:20970138 | pubmed:author | pubmed-author:BeltonOrinaO | lld:pubmed |
| pubmed-article:20970138 | pubmed:author | pubmed-author:O'GaoraPeadar... | lld:pubmed |
| pubmed-article:20970138 | pubmed:author | pubmed-author:McCarthyCatha... | lld:pubmed |
| pubmed-article:20970138 | pubmed:author | pubmed-author:McClellandSar... | lld:pubmed |
| pubmed-article:20970138 | pubmed:author | pubmed-author:de... | lld:pubmed |
| pubmed-article:20970138 | pubmed:author | pubmed-author:JamesWilliam... | lld:pubmed |
| pubmed-article:20970138 | pubmed:copyrightInfo | Copyright © 2010 Elsevier Ireland Ltd. All rights reserved. | lld:pubmed |
| pubmed-article:20970138 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20970138 | pubmed:volume | 213 | lld:pubmed |
| pubmed-article:20970138 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20970138 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20970138 | pubmed:pagination | 400-7 | lld:pubmed |
| pubmed-article:20970138 | pubmed:dateRevised | 2011-6-22 | lld:pubmed |
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| pubmed-article:20970138 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20970138 | pubmed:articleTitle | SorLA modulates atheroprotective properties of CLA by regulating monocyte migration. | lld:pubmed |
| pubmed-article:20970138 | pubmed:affiliation | School of Biomedical and Biomolecular Science, UCD Conway Institute, University College Dublin, Belfield, Dublin 4, Ireland. c.mccarthy@ucd.ie | lld:pubmed |
| pubmed-article:20970138 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20970138 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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