Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2010-10-12
pubmed:abstractText
The aim of this study was to investigate the mechanisms by which nicotine increases vascular smooth muscle cell (VSMC) proliferation and post-injury neointimal formation.
pubmed:grant
pubmed:commentsCorrections
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/Chelating Agents, http://linkedlifedata.com/resource/pubmed/chemical/Early Growth Response Protein 1, http://linkedlifedata.com/resource/pubmed/chemical/Egr1 protein, rat, http://linkedlifedata.com/resource/pubmed/chemical/Elk1 protein, rat, http://linkedlifedata.com/resource/pubmed/chemical/Ki-67 Antigen, http://linkedlifedata.com/resource/pubmed/chemical/Mitogen-Activated Protein Kinase 1, http://linkedlifedata.com/resource/pubmed/chemical/Mitogen-Activated Protein Kinase 3, http://linkedlifedata.com/resource/pubmed/chemical/Nicotine, http://linkedlifedata.com/resource/pubmed/chemical/Nicotinic Agonists, http://linkedlifedata.com/resource/pubmed/chemical/Nicotinic Antagonists, http://linkedlifedata.com/resource/pubmed/chemical/Protein Kinase Inhibitors, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Nicotinic, http://linkedlifedata.com/resource/pubmed/chemical/ets-Domain Protein Elk-1
pubmed:status
MEDLINE
pubmed:month
Nov
pubmed:issn
1755-3245
pubmed:author
pubmed:issnType
Electronic
pubmed:day
1
pubmed:volume
88
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
296-303
pubmed:dateRevised
2011-11-1
pubmed:meshHeading
pubmed-meshheading:20615913-Administration, Oral, pubmed-meshheading:20615913-Animals, pubmed-meshheading:20615913-Atherosclerosis, pubmed-meshheading:20615913-Balloon Dilation, pubmed-meshheading:20615913-Calcium Signaling, pubmed-meshheading:20615913-Cell Proliferation, pubmed-meshheading:20615913-Cells, Cultured, pubmed-meshheading:20615913-Chelating Agents, pubmed-meshheading:20615913-Disease Models, Animal, pubmed-meshheading:20615913-Dose-Response Relationship, Drug, pubmed-meshheading:20615913-Early Growth Response Protein 1, pubmed-meshheading:20615913-Enzyme Activation, pubmed-meshheading:20615913-Iliac Artery, pubmed-meshheading:20615913-Ki-67 Antigen, pubmed-meshheading:20615913-Mitogen-Activated Protein Kinase 1, pubmed-meshheading:20615913-Mitogen-Activated Protein Kinase 3, pubmed-meshheading:20615913-Muscle, Smooth, Vascular, pubmed-meshheading:20615913-Myocytes, Smooth Muscle, pubmed-meshheading:20615913-Nicotine, pubmed-meshheading:20615913-Nicotinic Agonists, pubmed-meshheading:20615913-Nicotinic Antagonists, pubmed-meshheading:20615913-Phosphorylation, pubmed-meshheading:20615913-Protein Kinase Inhibitors, pubmed-meshheading:20615913-RNA Interference, pubmed-meshheading:20615913-Rats, pubmed-meshheading:20615913-Rats, Sprague-Dawley, pubmed-meshheading:20615913-Receptors, Nicotinic, pubmed-meshheading:20615913-Time Factors, pubmed-meshheading:20615913-Transfection, pubmed-meshheading:20615913-Tunica Intima, pubmed-meshheading:20615913-ets-Domain Protein Elk-1
pubmed:year
2010
pubmed:articleTitle
Novel role of Egr-1 in nicotine-related neointimal formation.
pubmed:affiliation
Department of Surgery and Vascular Biology Institute, University of Miami Miller School of Medicine, 1600 NW 10th Avenue, RMSB 7147A, Miami, FL 33136, USA.
pubmed:publicationType
Journal Article, Research Support, N.I.H., Extramural