rdf:type |
|
lifeskim:mentions |
umls-concept:C0014257,
umls-concept:C0020507,
umls-concept:C0023175,
umls-concept:C0079633,
umls-concept:C0220781,
umls-concept:C0221464,
umls-concept:C0332282,
umls-concept:C0521447,
umls-concept:C1135918,
umls-concept:C1269955,
umls-concept:C1516960,
umls-concept:C1521761,
umls-concept:C1880177,
umls-concept:C1883254,
umls-concept:C2699153
|
pubmed:issue |
9
|
pubmed:dateCreated |
2010-8-19
|
pubmed:abstractText |
To validate the hypothesis that the toxic heavy metal lead (Pb) may be linked to cardiovascular diseases via the initiation of atherosclerosis, in vivo and in vitro studies were conducted.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Sep
|
pubmed:issn |
1524-4636
|
pubmed:author |
pubmed-author:BernhardDavidD,
pubmed-author:BlunderStefanS,
pubmed-author:DebbagePaulP,
pubmed-author:FrotschnigSandraS,
pubmed-author:KiechlStefanS,
pubmed-author:KnoflachMichaelM,
pubmed-author:KreutmayerSimone BSB,
pubmed-author:LauferGüntherG,
pubmed-author:MessnerBarbaraB,
pubmed-author:SeubertAndreasA,
pubmed-author:StelzmüllerMarlies EME,
pubmed-author:WallnoeferEvelynE,
pubmed-author:WickGeorgG,
pubmed-author:WilleitJohannJ,
pubmed-author:ZellerIrisI
|
pubmed:issnType |
Electronic
|
pubmed:volume |
30
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
1733-40
|
pubmed:meshHeading |
pubmed-meshheading:20595649-Adolescent,
pubmed-meshheading:20595649-Carotid Artery Diseases,
pubmed-meshheading:20595649-Cell Movement,
pubmed-meshheading:20595649-Cell Proliferation,
pubmed-meshheading:20595649-Cells, Cultured,
pubmed-meshheading:20595649-Dose-Response Relationship, Drug,
pubmed-meshheading:20595649-Elastin,
pubmed-meshheading:20595649-Endothelial Cells,
pubmed-meshheading:20595649-Female,
pubmed-meshheading:20595649-Heat-Shock Proteins,
pubmed-meshheading:20595649-Humans,
pubmed-meshheading:20595649-Hyperplasia,
pubmed-meshheading:20595649-Interleukin-8,
pubmed-meshheading:20595649-Lead,
pubmed-meshheading:20595649-Logistic Models,
pubmed-meshheading:20595649-Mammary Arteries,
pubmed-meshheading:20595649-Muscle, Smooth, Vascular,
pubmed-meshheading:20595649-Myocytes, Smooth Muscle,
pubmed-meshheading:20595649-NF-E2-Related Factor 2,
pubmed-meshheading:20595649-Odds Ratio,
pubmed-meshheading:20595649-Organ Culture Techniques,
pubmed-meshheading:20595649-Radial Artery,
pubmed-meshheading:20595649-Risk Assessment,
pubmed-meshheading:20595649-Risk Factors,
pubmed-meshheading:20595649-Severity of Illness Index,
pubmed-meshheading:20595649-Time Factors,
pubmed-meshheading:20595649-Tunica Intima,
pubmed-meshheading:20595649-Up-Regulation,
pubmed-meshheading:20595649-Young Adult
|
pubmed:year |
2010
|
pubmed:articleTitle |
Lead contributes to arterial intimal hyperplasia through nuclear factor erythroid 2-related factor-mediated endothelial interleukin 8 synthesis and subsequent invasion of smooth muscle cells.
|
pubmed:affiliation |
Cardiac Surgery, Research Laboratories, Department of Surgery, Medical University of Vienna, Vienna, Austria.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|