| pubmed-article:20433755 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20433755 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:20433755 | lifeskim:mentions | umls-concept:C1882071 | lld:lifeskim |
| pubmed-article:20433755 | lifeskim:mentions | umls-concept:C0529535 | lld:lifeskim |
| pubmed-article:20433755 | lifeskim:mentions | umls-concept:C0007102 | lld:lifeskim |
| pubmed-article:20433755 | lifeskim:mentions | umls-concept:C0334227 | lld:lifeskim |
| pubmed-article:20433755 | lifeskim:mentions | umls-concept:C0016360 | lld:lifeskim |
| pubmed-article:20433755 | lifeskim:mentions | umls-concept:C0079904 | lld:lifeskim |
| pubmed-article:20433755 | lifeskim:mentions | umls-concept:C1101610 | lld:lifeskim |
| pubmed-article:20433755 | lifeskim:mentions | umls-concept:C0680727 | lld:lifeskim |
| pubmed-article:20433755 | lifeskim:mentions | umls-concept:C1335191 | lld:lifeskim |
| pubmed-article:20433755 | pubmed:dateCreated | 2010-6-14 | lld:pubmed |
| pubmed-article:20433755 | pubmed:abstractText | Diminished expression or activity of prostate apoptosis response protein 4 (Par-4) has been demonstrated in a number of cancers, although reports on Par-4 expression during colon cancer progression are lacking. An understanding of the molecular events in conjunction with the genetic networks affected by Par-4 is warranted. | lld:pubmed |
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| pubmed-article:20433755 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:20433755 | pubmed:citationSubset | IM | lld:pubmed |
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