20300303

Source:http://linkedlifedata.com/resource/pubmed/id/20300303

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0008059, umls-concept:C0011854, umls-concept:C0332281, umls-concept:C0332307, umls-concept:C0337801, umls-concept:C0598095, umls-concept:C1332802
pubmed:issue	3
pubmed:dateCreated	2010-3-19
pubmed:abstractText	Type 1 diabetes (T1D) is an organ-specific autoimmune disease characterized by T cell-mediated destruction of pancreatic islets. T cell proliferation is negatively regulated by cytotoxic lymphocyte antigen-4 (CTLA-4). CTLA-4 polymorphisms are associated with T1D in some but not all populations.
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pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/101223637
pubmed:status	PubMed-not-MEDLINE
pubmed:month	Sep
pubmed:issn	0971-6866
pubmed:author	pubmed-author:LeskiMichaelM, pubmed-author:RohowskyNestorN, pubmed-author:SalehHatem MohamedHM
pubmed:issnType	Print
pubmed:volume	14
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	92-8
pubmed:year	2008
pubmed:articleTitle	The CTLA4 -819 C/T and +49 A/G dimorphisms are associated with Type 1 diabetes in Egyptian children.
pubmed:affiliation	Genotyping Lab for Genetics of Type One Diabetes- Autoimmune Diseases Section, R and D Department, The Egyptian Organization for Biotechnology (EGYTEC) - The Egyptian Organization for Biological Products and Vaccines (EGYVAC-VACSERA), 1 El Batal Ahmed Abdel Aziz Street, Agouza, Cairo 1312, Egypt.
pubmed:publicationType	Journal Article