Source:http://linkedlifedata.com/resource/pubmed/id/20180412
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
12
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pubmed:dateCreated |
2010-2-25
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pubmed:abstractText |
To investigate targeted degradation of the androgen receptor (AR) by chimeric molecules (DHT-PROTAC) via the ubiquitin-proteasome pathway in androgen-independent prostate cancer CA-2B cells, and explore the proliferation, secretion and apoptosis of the treated cells.
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pubmed:language |
chi
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
1009-3591
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
15
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1059-63
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pubmed:meshHeading |
pubmed-meshheading:20180412-Antineoplastic Agents,
pubmed-meshheading:20180412-Apoptosis,
pubmed-meshheading:20180412-Cell Line, Tumor,
pubmed-meshheading:20180412-Cell Proliferation,
pubmed-meshheading:20180412-Humans,
pubmed-meshheading:20180412-Male,
pubmed-meshheading:20180412-Prostate-Specific Antigen,
pubmed-meshheading:20180412-Prostatic Neoplasms,
pubmed-meshheading:20180412-Receptors, Androgen
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pubmed:year |
2009
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pubmed:articleTitle |
[Targeted degradation of androgen receptors in androgen-independent prostate cancer cells: an experimental study].
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pubmed:affiliation |
Department of Urology, The First People's Hospital Afiliated to Shanghai Jiaotong University, Shanghai 200080, China.
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pubmed:publicationType |
Journal Article,
English Abstract,
Research Support, Non-U.S. Gov't
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