pubmed-article:20051879 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20051879 | lifeskim:mentions | umls-concept:C0014257 | lld:lifeskim |
pubmed-article:20051879 | lifeskim:mentions | umls-concept:C0055538 | lld:lifeskim |
pubmed-article:20051879 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
pubmed-article:20051879 | lifeskim:mentions | umls-concept:C1514548 | lld:lifeskim |
pubmed-article:20051879 | lifeskim:mentions | umls-concept:C1450533 | lld:lifeskim |
pubmed-article:20051879 | lifeskim:mentions | umls-concept:C0332291 | lld:lifeskim |
pubmed-article:20051879 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:20051879 | pubmed:dateCreated | 2010-5-21 | lld:pubmed |
pubmed-article:20051879 | pubmed:abstractText | Torcetrapib, a prototype cholesteryl ester transfer protein (CETP) inhibitor with potential for decreasing atherosclerotic disease, increased cardiovascular events in clinical trials. The identified hypertensive and aldosterone-elevating actions of torcetrapib may not fully account for this elevated cardiovascular risk. Therefore, we evaluated the effects of torcetrapib on endothelial mediated vasodilation in vivo. | lld:pubmed |
pubmed-article:20051879 | pubmed:language | eng | lld:pubmed |
pubmed-article:20051879 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20051879 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:20051879 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20051879 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20051879 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20051879 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20051879 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20051879 | pubmed:month | May | lld:pubmed |
pubmed-article:20051879 | pubmed:issn | 1533-4023 | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:HuangZhihongZ | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:SarichTroy... | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:ParryTom JTJ | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:KuoGee-HongGH | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:ConnellyMarge... | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:DemarestKeith... | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:DamianoBruce... | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:ChenCailinC | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:CoolsFrankF | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:GiardinoEdwar... | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:CheungWai-Man... | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:GallacherDavi... | lld:pubmed |
pubmed-article:20051879 | pubmed:author | pubmed-author:Van der... | lld:pubmed |
pubmed-article:20051879 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20051879 | pubmed:volume | 55 | lld:pubmed |
pubmed-article:20051879 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20051879 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20051879 | pubmed:pagination | 459-68 | lld:pubmed |
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pubmed-article:20051879 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20051879 | pubmed:articleTitle | Torcetrapib produces endothelial dysfunction independent of cholesteryl ester transfer protein inhibition. | lld:pubmed |
pubmed-article:20051879 | pubmed:affiliation | Johnson & Johnson Pharmaceutical Research and Development, Welsh & McKean Roads, Spring House, PA 19477-0776, USA. mconnell@its.jnj.com | lld:pubmed |
pubmed-article:20051879 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:20051879 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |