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PredicateObject
rdf:type
lifeskim:mentions
pubmed:dateCreated
2010-1-12
pubmed:abstractText
Neuroinflammation occurs after seizures and is implicated in epileptogenesis. CCR2 is a chemokine receptor for CCL2 and their interaction mediates monocyte infiltration in the neuroinflammatory cascade triggered in different brain pathologies. In this work CCR2 and CCL2 expression were examined following status epilepticus (SE) induced by pilocarpine injection.
pubmed:grant
pubmed:commentsCorrections
http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-10190688, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-10430051, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-10559781, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-10828541, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-10993690, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-11063828, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-11152700, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-12495638, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-12687707, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-14576355, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-14580952, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-14581618, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-14732474, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-15804429, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-16025454, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-16196033, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-16302852, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-16344154, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-16480853, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-16554469, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-16886201, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-17183554, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-17191078, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-1793176, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-17949991, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-18226170, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-18375756, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-18402383, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-18461605, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-18637012, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-18796338, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-2926331, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-4110397, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-7694849, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-7805752, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-8170963, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-8872898, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-9342361, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-9362535, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-9463410, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-9501225, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-9628469, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-9655469, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-9732872, http://linkedlifedata.com/resource/pubmed/commentcorrection/20034406-9818943
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
1742-2094
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
6
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
40
pubmed:dateRevised
2010-9-28
pubmed:meshHeading
pubmed-meshheading:20034406-Animals, pubmed-meshheading:20034406-Astrocytes, pubmed-meshheading:20034406-CA1 Region, Hippocampal, pubmed-meshheading:20034406-Cell Count, pubmed-meshheading:20034406-Chemokine CCL2, pubmed-meshheading:20034406-Dentate Gyrus, pubmed-meshheading:20034406-Endothelial Cells, pubmed-meshheading:20034406-Hippocampus, pubmed-meshheading:20034406-Immunohistochemistry, pubmed-meshheading:20034406-Microglia, pubmed-meshheading:20034406-Microscopy, Confocal, pubmed-meshheading:20034406-Neuroglia, pubmed-meshheading:20034406-Neurons, pubmed-meshheading:20034406-Pilocarpine, pubmed-meshheading:20034406-Rats, pubmed-meshheading:20034406-Rats, Sprague-Dawley, pubmed-meshheading:20034406-Receptors, CCR2, pubmed-meshheading:20034406-Seizures, pubmed-meshheading:20034406-Status Epilepticus
pubmed:year
2009
pubmed:articleTitle
Chemokine CCL2 and its receptor CCR2 are increased in the hippocampus following pilocarpine-induced status epilepticus.
pubmed:affiliation
Department of Neurosurgery, Scott and White Hospital, Temple, TX 76503, USA. mforesti@swmail.sw.org
pubmed:publicationType
Journal Article
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