rdf:type |
|
lifeskim:mentions |
umls-concept:C0017262,
umls-concept:C0031437,
umls-concept:C0036525,
umls-concept:C0086418,
umls-concept:C0185117,
umls-concept:C0439849,
umls-concept:C0445223,
umls-concept:C0684249,
umls-concept:C1522484,
umls-concept:C1552599,
umls-concept:C1704787,
umls-concept:C2911684
|
pubmed:issue |
8
|
pubmed:dateCreated |
2009-10-6
|
pubmed:abstractText |
A strong link has been recently demonstrated between inflammation and lung cancer. Thus, we investigated whether the proinflammatory cytokine IL-32 may be involved in lung carcinogenesis and hence provide a novel therapeutic target.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Oct
|
pubmed:issn |
1535-4970
|
pubmed:author |
|
pubmed:issnType |
Electronic
|
pubmed:day |
15
|
pubmed:volume |
180
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
769-79
|
pubmed:meshHeading |
|
pubmed:year |
2009
|
pubmed:articleTitle |
Expression of IL-32 in human lung cancer is related to the histotype and metastatic phenotype.
|
pubmed:affiliation |
Department of Oncology and Neurosciences, Anatomic Pathology Section, G. d'Annunzio University, Italy. .
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|