| pubmed-article:19596916 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19596916 | lifeskim:mentions | umls-concept:C0007102 | lld:lifeskim |
| pubmed-article:19596916 | lifeskim:mentions | umls-concept:C0031715 | lld:lifeskim |
| pubmed-article:19596916 | lifeskim:mentions | umls-concept:C0334227 | lld:lifeskim |
| pubmed-article:19596916 | lifeskim:mentions | umls-concept:C1411976 | lld:lifeskim |
| pubmed-article:19596916 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
| pubmed-article:19596916 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:19596916 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:19596916 | pubmed:dateCreated | 2009-7-14 | lld:pubmed |
| pubmed-article:19596916 | pubmed:abstractText | Toll-like receptors (TLRs) are involved in innate immunity. Overexpression of TLRs has been implicated in various types of cancer including colorectal cancer (CRC). The phosphatidylinositol-3'-kinase (PI3K)/Akt signaling pathway is involved in CRC growth and progression. In this study, we determined whether TLR4 signaling and PI3K/Akt pathway activation occur in CRCs. | lld:pubmed |
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| pubmed-article:19596916 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19596916 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19596916 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19596916 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19596916 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:19596916 | pubmed:issn | 1791-7530 | lld:pubmed |
| pubmed-article:19596916 | pubmed:author | pubmed-author:EversB MarkBM | lld:pubmed |
| pubmed-article:19596916 | pubmed:author | pubmed-author:BowenKanika... | lld:pubmed |
| pubmed-article:19596916 | pubmed:author | pubmed-author:DoanHung QHQ | lld:pubmed |
| pubmed-article:19596916 | pubmed:author | pubmed-author:JacksonLindse... | lld:pubmed |
| pubmed-article:19596916 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19596916 | pubmed:volume | 29 | lld:pubmed |
| pubmed-article:19596916 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19596916 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19596916 | pubmed:pagination | 2473-8 | lld:pubmed |
| pubmed-article:19596916 | pubmed:dateRevised | 2011-4-13 | lld:pubmed |
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| pubmed-article:19596916 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19596916 | pubmed:articleTitle | Toll-like receptor 4 activation increases Akt phosphorylation in colon cancer cells. | lld:pubmed |
| pubmed-article:19596916 | pubmed:affiliation | Department of Surgery, The University of Texas Medical Branch, Galveston, TX, USA. | lld:pubmed |
| pubmed-article:19596916 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19596916 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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