| pubmed-article:19546226 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19546226 | lifeskim:mentions | umls-concept:C0014939 | lld:lifeskim |
| pubmed-article:19546226 | lifeskim:mentions | umls-concept:C0596290 | lld:lifeskim |
| pubmed-article:19546226 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
| pubmed-article:19546226 | lifeskim:mentions | umls-concept:C1709059 | lld:lifeskim |
| pubmed-article:19546226 | pubmed:issue | 35 | lld:pubmed |
| pubmed-article:19546226 | pubmed:dateCreated | 2009-8-24 | lld:pubmed |
| pubmed-article:19546226 | pubmed:abstractText | High expression of the estrogen receptor-related receptor (ERR)-alpha in human tumors is correlated to a poor prognosis, suggesting an involvement of the receptor in cell proliferation. In this study, we show that a synthetic compound (XCT790) that modulates the activity of ERRalpha reduces the proliferation of various cell lines and blocks the G(1)/S transition of the cell cycle in an ERRalpha-dependent manner. XCT790 induces, in a p53-independent manner, the expression of the cell cycle inhibitor p21(waf/cip)(1) at the protein, mRNA, and promoter level, leading to an accumulation of hypophosphorylated Rb. Finally, XCT790 reduces cell tumorigenicity in Nude mice. | lld:pubmed |
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| pubmed-article:19546226 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19546226 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19546226 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:19546226 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:19546226 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19546226 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:19546226 | pubmed:issn | 0021-9258 | lld:pubmed |
| pubmed-article:19546226 | pubmed:author | pubmed-author:NorthSophieS | lld:pubmed |
| pubmed-article:19546226 | pubmed:author | pubmed-author:VanackerJean-... | lld:pubmed |
| pubmed-article:19546226 | pubmed:author | pubmed-author:LanvinOliviaO | lld:pubmed |
| pubmed-article:19546226 | pubmed:author | pubmed-author:TribolletViol... | lld:pubmed |
| pubmed-article:19546226 | pubmed:author | pubmed-author:BiancoStéphan... | lld:pubmed |
| pubmed-article:19546226 | pubmed:author | pubmed-author:MacariClaireC | lld:pubmed |
| pubmed-article:19546226 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:19546226 | pubmed:day | 28 | lld:pubmed |
| pubmed-article:19546226 | pubmed:volume | 284 | lld:pubmed |
| pubmed-article:19546226 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19546226 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19546226 | pubmed:pagination | 23286-92 | lld:pubmed |
| pubmed-article:19546226 | pubmed:dateRevised | 2010-9-27 | lld:pubmed |
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| pubmed-article:19546226 | pubmed:meshHeading | pubmed-meshheading:19546226... | lld:pubmed |
| pubmed-article:19546226 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19546226 | pubmed:articleTitle | Modulating estrogen receptor-related receptor-alpha activity inhibits cell proliferation. | lld:pubmed |
| pubmed-article:19546226 | pubmed:affiliation | Institut de Génomique Fonctionnelle de Lyon, Université de Lyon, Université Lyon 1, France. | lld:pubmed |
| pubmed-article:19546226 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19546226 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:2101 | entrezgene:pubmed | pubmed-article:19546226 | lld:entrezgene |
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