pubmed-article:18993115 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18993115 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:18993115 | lifeskim:mentions | umls-concept:C0225833 | lld:lifeskim |
pubmed-article:18993115 | lifeskim:mentions | umls-concept:C0003009 | lld:lifeskim |
pubmed-article:18993115 | lifeskim:mentions | umls-concept:C0597357 | lld:lifeskim |
pubmed-article:18993115 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:18993115 | lifeskim:mentions | umls-concept:C1412113 | lld:lifeskim |
pubmed-article:18993115 | lifeskim:mentions | umls-concept:C0439851 | lld:lifeskim |
pubmed-article:18993115 | lifeskim:mentions | umls-concept:C1552596 | lld:lifeskim |
pubmed-article:18993115 | lifeskim:mentions | umls-concept:C0332206 | lld:lifeskim |
pubmed-article:18993115 | lifeskim:mentions | umls-concept:C1947931 | lld:lifeskim |
pubmed-article:18993115 | pubmed:issue | 12 | lld:pubmed |
pubmed-article:18993115 | pubmed:dateCreated | 2008-11-28 | lld:pubmed |
pubmed-article:18993115 | pubmed:abstractText | Atrial fibrillation (AF) is the most common cardiac arrhythmia in clinical practice. Indirect evidence from clinical trials demonstrates that chronic inhibition of the renin-angiotensin-system (RAS) significantly reduces the incidence of AF. Since mechanisms of this protective effect of RAS-blockade are poorly understood, we directly tested proarrhythmic effects of angiotensin II (Ang II) in human atrial myocardium. | lld:pubmed |
pubmed-article:18993115 | pubmed:language | eng | lld:pubmed |
pubmed-article:18993115 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18993115 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:18993115 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18993115 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18993115 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18993115 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18993115 | pubmed:month | Dec | lld:pubmed |
pubmed-article:18993115 | pubmed:issn | 1388-9842 | lld:pubmed |
pubmed-article:18993115 | pubmed:author | pubmed-author:HasenfussGerd... | lld:pubmed |
pubmed-article:18993115 | pubmed:author | pubmed-author:PieskeBurkert... | lld:pubmed |
pubmed-article:18993115 | pubmed:author | pubmed-author:SchöndubeFrie... | lld:pubmed |
pubmed-article:18993115 | pubmed:author | pubmed-author:KockskämperJe... | lld:pubmed |
pubmed-article:18993115 | pubmed:author | pubmed-author:von... | lld:pubmed |
pubmed-article:18993115 | pubmed:author | pubmed-author:ZhuDananD | lld:pubmed |
pubmed-article:18993115 | pubmed:author | pubmed-author:SchmittoJan... | lld:pubmed |
pubmed-article:18993115 | pubmed:author | pubmed-author:RübertusSven-... | lld:pubmed |
pubmed-article:18993115 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:18993115 | pubmed:volume | 10 | lld:pubmed |
pubmed-article:18993115 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18993115 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18993115 | pubmed:pagination | 1172-6 | lld:pubmed |
pubmed-article:18993115 | pubmed:dateRevised | 2011-6-8 | lld:pubmed |
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pubmed-article:18993115 | pubmed:meshHeading | pubmed-meshheading:18993115... | lld:pubmed |
pubmed-article:18993115 | pubmed:year | 2008 | lld:pubmed |
pubmed-article:18993115 | pubmed:articleTitle | Direct pro-arrhythmogenic effects of angiotensin II can be suppressed by AT1 receptor blockade in human atrial myocardium. | lld:pubmed |
pubmed-article:18993115 | pubmed:affiliation | Department of Cardiology, Medical University Graz, Graz, Austria. dirk.von-lewinski@meduni-graz.at | lld:pubmed |
pubmed-article:18993115 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:18993115 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:18993115 | lld:pubmed |