| pubmed-article:18658095 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18658095 | lifeskim:mentions | umls-concept:C0058402 | lld:lifeskim |
| pubmed-article:18658095 | lifeskim:mentions | umls-concept:C1704816 | lld:lifeskim |
| pubmed-article:18658095 | lifeskim:mentions | umls-concept:C0682972 | lld:lifeskim |
| pubmed-article:18658095 | lifeskim:mentions | umls-concept:C1414313 | lld:lifeskim |
| pubmed-article:18658095 | lifeskim:mentions | umls-concept:C1366765 | lld:lifeskim |
| pubmed-article:18658095 | lifeskim:mentions | umls-concept:C1412186 | lld:lifeskim |
| pubmed-article:18658095 | lifeskim:mentions | umls-concept:C0166825 | lld:lifeskim |
| pubmed-article:18658095 | lifeskim:mentions | umls-concept:C0086597 | lld:lifeskim |
| pubmed-article:18658095 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:18658095 | lifeskim:mentions | umls-concept:C1998811 | lld:lifeskim |
| pubmed-article:18658095 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:18658095 | pubmed:dateCreated | 2009-1-6 | lld:pubmed |
| pubmed-article:18658095 | pubmed:abstractText | Previous studies have shown that wounding of human corneal epithelial cells (HCECs) results in the release of G-protein-coupled receptor ligands such as ATP and lysophosphatidic acid (LPA), which in turn transactivate epidermal growth factor (EGF) receptor (EGFR) through ectodomain shedding of heparin-binding EGF-like growth factor (HB-EGF). In the present study, the role of extracellular signal-regulated kinases 1/2 (ERK1/2) in regulating EGFR transactivation was investigated. | lld:pubmed |
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| pubmed-article:18658095 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18658095 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18658095 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18658095 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:18658095 | pubmed:issn | 1552-5783 | lld:pubmed |
| pubmed-article:18658095 | pubmed:author | pubmed-author:LuiY CYC | lld:pubmed |
| pubmed-article:18658095 | pubmed:author | pubmed-author:YuFu-Shin XFS | lld:pubmed |
| pubmed-article:18658095 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18658095 | pubmed:volume | 50 | lld:pubmed |
| pubmed-article:18658095 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18658095 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18658095 | pubmed:pagination | 132-9 | lld:pubmed |
| pubmed-article:18658095 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:18658095 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:18658095 | pubmed:articleTitle | ERK1/2 mediate wounding- and G-protein-coupled receptor ligands-induced EGFR activation via regulating ADAM17 and HB-EGF shedding. | lld:pubmed |
| pubmed-article:18658095 | pubmed:affiliation | Department of Ophthalmology, Kresge Eye Institute, Wayne State University School of Medicine, 4717 St. Antoine Boulevard, Detroit, MI 48201, USA. | lld:pubmed |
| pubmed-article:18658095 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18658095 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:18658095 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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