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| pubmed-article:18559695 | lifeskim:mentions | umls-concept:C1704632 | lld:lifeskim |
| pubmed-article:18559695 | lifeskim:mentions | umls-concept:C0871261 | lld:lifeskim |
| pubmed-article:18559695 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
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| pubmed-article:18559695 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
| pubmed-article:18559695 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
| pubmed-article:18559695 | lifeskim:mentions | umls-concept:C0065055 | lld:lifeskim |
| pubmed-article:18559695 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:18559695 | pubmed:dateCreated | 2008-7-22 | lld:pubmed |
| pubmed-article:18559695 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18559695 | pubmed:abstractText | HMGCR(3-Hydroxy-3-methylglutaryl coenzyme A reductase), the direct target of statin inhibition, undergoes alternative splicing of exon 13, which encodes part of the statin-binding domain of the enzyme. We hypothesized that HMGCR alternative splicing might be related to the interindividual variation in plasma low-density lipoprotein cholesterol response to statin treatment. | lld:pubmed |
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| pubmed-article:18559695 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18559695 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:18559695 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18559695 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:18559695 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:18559695 | pubmed:author | pubmed-author:GaoFengF | lld:pubmed |
| pubmed-article:18559695 | pubmed:author | pubmed-author:RotterJerome... | lld:pubmed |
| pubmed-article:18559695 | pubmed:author | pubmed-author:KraussRonald... | lld:pubmed |
| pubmed-article:18559695 | pubmed:author | pubmed-author:RuanWeimingW | lld:pubmed |
| pubmed-article:18559695 | pubmed:author | pubmed-author:MedinaMarisa... | lld:pubmed |
| pubmed-article:18559695 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18559695 | pubmed:day | 22 | lld:pubmed |
| pubmed-article:18559695 | pubmed:volume | 118 | lld:pubmed |
| pubmed-article:18559695 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18559695 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18559695 | pubmed:pagination | 355-62 | lld:pubmed |
| pubmed-article:18559695 | pubmed:dateRevised | 2010-10-4 | lld:pubmed |
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| pubmed-article:18559695 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18559695 | pubmed:articleTitle | Alternative splicing of 3-hydroxy-3-methylglutaryl coenzyme A reductase is associated with plasma low-density lipoprotein cholesterol response to simvastatin. | lld:pubmed |
| pubmed-article:18559695 | pubmed:affiliation | Children's Hospital Oakland Research Institute, CA 94609, USA. | lld:pubmed |
| pubmed-article:18559695 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18559695 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:18559695 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
