pubmed-article:18212286 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18212286 | lifeskim:mentions | umls-concept:C1522564 | lld:lifeskim |
pubmed-article:18212286 | lifeskim:mentions | umls-concept:C0392747 | lld:lifeskim |
pubmed-article:18212286 | lifeskim:mentions | umls-concept:C0302189 | lld:lifeskim |
pubmed-article:18212286 | lifeskim:mentions | umls-concept:C1710236 | lld:lifeskim |
pubmed-article:18212286 | lifeskim:mentions | umls-concept:C1554963 | lld:lifeskim |
pubmed-article:18212286 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:18212286 | pubmed:dateCreated | 2008-2-12 | lld:pubmed |
pubmed-article:18212286 | pubmed:abstractText | Traditional antiarrhythmic pharmacological therapies are limited by their global cardiac action, low efficacy, and significant proarrhythmic effects. We present a novel approach for the modification of the myocardial electrophysiological substrate using cell grafts genetically engineered to express specific ionic channels. | lld:pubmed |
pubmed-article:18212286 | pubmed:language | eng | lld:pubmed |
pubmed-article:18212286 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18212286 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:18212286 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18212286 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18212286 | pubmed:month | Feb | lld:pubmed |
pubmed-article:18212286 | pubmed:issn | 1524-4539 | lld:pubmed |
pubmed-article:18212286 | pubmed:author | pubmed-author:FeldYairY | lld:pubmed |
pubmed-article:18212286 | pubmed:author | pubmed-author:MaromShimonS | lld:pubmed |
pubmed-article:18212286 | pubmed:author | pubmed-author:GepsteinLiorL | lld:pubmed |
pubmed-article:18212286 | pubmed:author | pubmed-author:AronsonDoronD | lld:pubmed |
pubmed-article:18212286 | pubmed:author | pubmed-author:HuberIritI | lld:pubmed |
pubmed-article:18212286 | pubmed:author | pubmed-author:GepsteinAmira... | lld:pubmed |
pubmed-article:18212286 | pubmed:author | pubmed-author:YankelsonLior... | lld:pubmed |
pubmed-article:18212286 | pubmed:author | pubmed-author:ItzhakiIlanit... | lld:pubmed |
pubmed-article:18212286 | pubmed:author | pubmed-author:Bressler-Stra... | lld:pubmed |
pubmed-article:18212286 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:18212286 | pubmed:day | 12 | lld:pubmed |
pubmed-article:18212286 | pubmed:volume | 117 | lld:pubmed |
pubmed-article:18212286 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18212286 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18212286 | pubmed:pagination | 720-31 | lld:pubmed |
pubmed-article:18212286 | pubmed:meshHeading | pubmed-meshheading:18212286... | lld:pubmed |
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pubmed-article:18212286 | pubmed:meshHeading | pubmed-meshheading:18212286... | lld:pubmed |
pubmed-article:18212286 | pubmed:year | 2008 | lld:pubmed |
pubmed-article:18212286 | pubmed:articleTitle | Cell therapy for modification of the myocardial electrophysiological substrate. | lld:pubmed |
pubmed-article:18212286 | pubmed:affiliation | Sohnis Laboratory for Cardiac Electrophysiology and Regenerative Medicine, Bruce Rappaport Faculty of Medicine, Technion-Israel Institute of Technology, Haifa, Israel. | lld:pubmed |
pubmed-article:18212286 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:18212286 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |