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pubmed-article:17622738pubmed:abstractTextThe angiotensin II level is elevated in subjects genetically prone to develop hypertension, triggering renal hypercellularity, cytokine production, and matrix deposition. Angiotensin-converting enzyme inhibition and/or angiotensin II type 1 receptor blockade attenuate renal damage. Rosiglitazone, a peroxisome proliferator-activated receptor gamma agonist possessing antihypertensive and anti-inflammatory properties, was demonstrated to provide better renal protection than angiotensin-converting enzyme inhibitors. We studied the effects of in vivo peroxisome proliferator-activated receptor gamma activation by rosiglitazone on angiotensin II synthesis, proliferation, and apoptosis of mesangial cells of spontaneously hypertensive rats versus normotensive Sprague-Dawley rats.lld:pubmed
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pubmed-article:17622738pubmed:copyrightInfoCopyright 2007 S. Karger AG, Basel.lld:pubmed
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pubmed-article:17622738pubmed:articleTitlePPAR-gamma activation inhibits angiotensin II synthesis, apoptosis, and proliferation of mesangial cells from spontaneously hypertensive rats.lld:pubmed
pubmed-article:17622738pubmed:affiliationNephrology Division, Department of Nephrology, Assaf Harofeh Medical Center, Zerifin, Israel. efratishai@013.netlld:pubmed
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