| pubmed-article:17206701 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17206701 | lifeskim:mentions | umls-concept:C0314921 | lld:lifeskim |
| pubmed-article:17206701 | lifeskim:mentions | umls-concept:C0009319 | lld:lifeskim |
| pubmed-article:17206701 | lifeskim:mentions | umls-concept:C2350483 | lld:lifeskim |
| pubmed-article:17206701 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
| pubmed-article:17206701 | lifeskim:mentions | umls-concept:C0019740 | lld:lifeskim |
| pubmed-article:17206701 | lifeskim:mentions | umls-concept:C1332714 | lld:lifeskim |
| pubmed-article:17206701 | lifeskim:mentions | umls-concept:C0086597 | lld:lifeskim |
| pubmed-article:17206701 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:17206701 | pubmed:dateCreated | 2007-5-25 | lld:pubmed |
| pubmed-article:17206701 | pubmed:abstractText | HLA-B27/beta2 microglobulin transgenic (TG) rats develop spontaneous colitis when raised under specific pathogen-free (SPF) conditions or after mono-association with Bacteroides vulgatus (B. vulgatus), whereas germ-free TG rats fail to develop intestinal inflammation. SPF HLA-B27 TG rnu/rnu rats, which are congenitally athymic, remain disease free. These results indicate that commensal intestinal bacteria and T cells are both pivotal for the development of colitis in TG rats. However, it is not known if T cells are also required in the induction of colitis by a single bacterial strain. The aim of this study was therefore to investigate the role of T cells in the development of colitis in B. vulgatus-monoassociated HLA-B27 TG rats. | lld:pubmed |
| pubmed-article:17206701 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17206701 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17206701 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17206701 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17206701 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17206701 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:17206701 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17206701 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17206701 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17206701 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17206701 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:17206701 | pubmed:issn | 1078-0998 | lld:pubmed |
| pubmed-article:17206701 | pubmed:author | pubmed-author:XuN ZNZ | lld:pubmed |
| pubmed-article:17206701 | pubmed:author | pubmed-author:DielemanLevin... | lld:pubmed |
| pubmed-article:17206701 | pubmed:author | pubmed-author:TonkonogySusa... | lld:pubmed |
| pubmed-article:17206701 | pubmed:author | pubmed-author:SartorR... | lld:pubmed |
| pubmed-article:17206701 | pubmed:author | pubmed-author:HoentjenFrank... | lld:pubmed |
| pubmed-article:17206701 | pubmed:author | pubmed-author:QianBi-FengBF | lld:pubmed |
| pubmed-article:17206701 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17206701 | pubmed:volume | 13 | lld:pubmed |
| pubmed-article:17206701 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17206701 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17206701 | pubmed:pagination | 317-24 | lld:pubmed |
| pubmed-article:17206701 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
| pubmed-article:17206701 | pubmed:meshHeading | pubmed-meshheading:17206701... | lld:pubmed |
| pubmed-article:17206701 | pubmed:meshHeading | pubmed-meshheading:17206701... | lld:pubmed |
| pubmed-article:17206701 | pubmed:meshHeading | pubmed-meshheading:17206701... | lld:pubmed |
| pubmed-article:17206701 | pubmed:meshHeading | pubmed-meshheading:17206701... | lld:pubmed |
| pubmed-article:17206701 | pubmed:meshHeading | pubmed-meshheading:17206701... | lld:pubmed |
| pubmed-article:17206701 | pubmed:meshHeading | pubmed-meshheading:17206701... | lld:pubmed |
| pubmed-article:17206701 | pubmed:meshHeading | pubmed-meshheading:17206701... | lld:pubmed |
| pubmed-article:17206701 | pubmed:meshHeading | pubmed-meshheading:17206701... | lld:pubmed |
| pubmed-article:17206701 | pubmed:meshHeading | pubmed-meshheading:17206701... | lld:pubmed |
| pubmed-article:17206701 | pubmed:meshHeading | pubmed-meshheading:17206701... | lld:pubmed |
| pubmed-article:17206701 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17206701 | pubmed:articleTitle | CD4(+) T lymphocytes mediate colitis in HLA-B27 transgenic rats monoassociated with nonpathogenic Bacteroides vulgatus. | lld:pubmed |
| pubmed-article:17206701 | pubmed:affiliation | Division of Gastroenterology and Hepatology, University of North Carolina, Chapel Hill, North Carolina, USA. | lld:pubmed |
| pubmed-article:17206701 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17206701 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:17206701 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:17206701 | lld:pubmed |
