Linked Life Data

17148555

Source:http://linkedlifedata.com/resource/pubmed/id/17148555

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2007-2-7
pubmed:abstractText
Polycystic ovary syndrome (PCOS), the most common cause of anovulatory infertility, is characterized by increased ovarian androgen production and arrested follicle development and is frequently associated with insulin resistance. These PCOS phenotypes are associated with exaggerated ovarian responsiveness to FSH and increased pregnancy loss.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
0021-972X
pubmed:author
pubmed:issnType
Print
pubmed:volume
92
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
705-13
pubmed:dateRevised
2008-11-21
pubmed:meshHeading
pubmed-meshheading:17148555-Base Sequence, pubmed-meshheading:17148555-Cell Cycle Proteins, pubmed-meshheading:17148555-Centrosome, pubmed-meshheading:17148555-Female, pubmed-meshheading:17148555-Gene Expression Profiling, pubmed-meshheading:17148555-Humans, pubmed-meshheading:17148555-Meiosis, pubmed-meshheading:17148555-Mitosis, pubmed-meshheading:17148555-Molecular Sequence Data, pubmed-meshheading:17148555-Oligonucleotide Array Sequence Analysis, pubmed-meshheading:17148555-Oocytes, pubmed-meshheading:17148555-PPAR gamma, pubmed-meshheading:17148555-Polycystic Ovary Syndrome, pubmed-meshheading:17148555-Promoter Regions, Genetic, pubmed-meshheading:17148555-Receptors, Androgen, pubmed-meshheading:17148555-Receptors, Cytoplasmic and Nuclear, pubmed-meshheading:17148555-Retinoid X Receptor alpha
pubmed:year
2007
pubmed:articleTitle
Molecular abnormalities in oocytes from women with polycystic ovary syndrome revealed by microarray analysis.
pubmed:affiliation
Department of Animal Science, University of Nebraska-Lincoln, 3800 Fair Street, Lincoln, Nebraska 68583-0908, USA. jwood5@unl.edu
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, Non-P.H.S., Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural