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| pubmed-article:16886896 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:16886896 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:16886896 | lifeskim:mentions | umls-concept:C0024623 | lld:lifeskim |
| pubmed-article:16886896 | lifeskim:mentions | umls-concept:C0555952 | lld:lifeskim |
| pubmed-article:16886896 | lifeskim:mentions | umls-concept:C0919534 | lld:lifeskim |
| pubmed-article:16886896 | lifeskim:mentions | umls-concept:C1979844 | lld:lifeskim |
| pubmed-article:16886896 | lifeskim:mentions | umls-concept:C0032529 | lld:lifeskim |
| pubmed-article:16886896 | lifeskim:mentions | umls-concept:C0439101 | lld:lifeskim |
| pubmed-article:16886896 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:16886896 | pubmed:dateCreated | 2006-8-4 | lld:pubmed |
| pubmed-article:16886896 | pubmed:abstractText | Glutathione S-transferases (GSTs) are considered to be cancer susceptibility genes as they play a role in the detoxification of carcinogenic species. This study aimed to elucidate the influence of several GST polymorphisms on colorectal and gastric cancer risk. | lld:pubmed |
| pubmed-article:16886896 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16886896 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16886896 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16886896 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16886896 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:16886896 | pubmed:issn | 1462-2416 | lld:pubmed |
| pubmed-article:16886896 | pubmed:author | pubmed-author:LaderoJosé... | lld:pubmed |
| pubmed-article:16886896 | pubmed:author | pubmed-author:Díaz-RubioMan... | lld:pubmed |
| pubmed-article:16886896 | pubmed:author | pubmed-author:MartínezCarme... | lld:pubmed |
| pubmed-article:16886896 | pubmed:author | pubmed-author:García-Martín... | lld:pubmed |
| pubmed-article:16886896 | pubmed:author | pubmed-author:SastreJavierJ | lld:pubmed |
| pubmed-article:16886896 | pubmed:author | pubmed-author:AgúndezJosé... | lld:pubmed |
| pubmed-article:16886896 | pubmed:author | pubmed-author:MartínFelipeF | lld:pubmed |
| pubmed-article:16886896 | pubmed:author | pubmed-author:FernándezJuan... | lld:pubmed |
| pubmed-article:16886896 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16886896 | pubmed:volume | 7 | lld:pubmed |
| pubmed-article:16886896 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16886896 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16886896 | pubmed:pagination | 711-8 | lld:pubmed |
| pubmed-article:16886896 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:16886896 | pubmed:meshHeading | pubmed-meshheading:16886896... | lld:pubmed |
| pubmed-article:16886896 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16886896 | pubmed:articleTitle | Glutathione S-transferases mu 1, theta 1, pi 1, alpha 1 and mu 3 genetic polymorphisms and the risk of colorectal and gastric cancers in humans. | lld:pubmed |
| pubmed-article:16886896 | pubmed:affiliation | Department of Pharmacology and Psychiatry, University of Extremadura, Badajoz, Spain. | lld:pubmed |
| pubmed-article:16886896 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16886896 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:16886896 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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