rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
2
|
pubmed:dateCreated |
2006-7-25
|
pubmed:abstractText |
Cardiopulmonary bypass (CPB) is known to cause cerebrovascular dysfunction. The etiology of these complications is complex, but disruption of normal cerebral endothelial function as a consequence of inflammatory or hypoperfusion phenomena have been implicated. The aim of this study was to investigate the effect of CPB time on cerebrovascular reactivity and to investigate the correlation of these findings with measured inflammatory markers.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Aug
|
pubmed:issn |
1552-6259
|
pubmed:author |
|
pubmed:issnType |
Electronic
|
pubmed:volume |
82
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
672-8
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:16863783-Acetylcholine,
pubmed-meshheading:16863783-Animals,
pubmed-meshheading:16863783-Cardiopulmonary Bypass,
pubmed-meshheading:16863783-Cerebral Arteries,
pubmed-meshheading:16863783-Endothelium, Vascular,
pubmed-meshheading:16863783-Immunohistochemistry,
pubmed-meshheading:16863783-Intercellular Adhesion Molecule-1,
pubmed-meshheading:16863783-Male,
pubmed-meshheading:16863783-Oxidative Stress,
pubmed-meshheading:16863783-Rats,
pubmed-meshheading:16863783-Rats, Sprague-Dawley,
pubmed-meshheading:16863783-Tumor Necrosis Factor-alpha,
pubmed-meshheading:16863783-Vasodilation
|
pubmed:year |
2006
|
pubmed:articleTitle |
Changes in cerebral vascular reactivity occur early during cardiopulmonary bypass in the rat.
|
pubmed:affiliation |
Service de Chirurgie Cardiovasculaire, Hôpital Cardiologique, Lille, France.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|