| pubmed-article:16551349 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16551349 | lifeskim:mentions | umls-concept:C0032659 | lld:lifeskim |
| pubmed-article:16551349 | lifeskim:mentions | umls-concept:C0038454 | lld:lifeskim |
| pubmed-article:16551349 | lifeskim:mentions | umls-concept:C0163401 | lld:lifeskim |
| pubmed-article:16551349 | lifeskim:mentions | umls-concept:C0052451 | lld:lifeskim |
| pubmed-article:16551349 | lifeskim:mentions | umls-concept:C0018591 | lld:lifeskim |
| pubmed-article:16551349 | lifeskim:mentions | umls-concept:C0678951 | lld:lifeskim |
| pubmed-article:16551349 | lifeskim:mentions | umls-concept:C0936012 | lld:lifeskim |
| pubmed-article:16551349 | pubmed:dateCreated | 2006-4-14 | lld:pubmed |
| pubmed-article:16551349 | pubmed:abstractText | Paraoxonase (PON) has anti-atherogenic activity due to its protective function against low density lipoprotein (LDL) oxidation. Alteration of enzyme activity due to polymorphisms in the PON genes may influence the development of atheroma and thus affect stroke risk. Three PON genes (PON1, PON2 and PON3) have been identifiedand mapped to chromosome 7. | lld:pubmed |
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| pubmed-article:16551349 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:16551349 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16551349 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16551349 | pubmed:issn | 1471-2350 | lld:pubmed |
| pubmed-article:16551349 | pubmed:author | pubmed-author:CheungJohnJ | lld:pubmed |
| pubmed-article:16551349 | pubmed:author | pubmed-author:St... | lld:pubmed |
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| pubmed-article:16551349 | pubmed:author | pubmed-author:WhalleyLawren... | lld:pubmed |
| pubmed-article:16551349 | pubmed:author | pubmed-author:PasdarAlireza... | lld:pubmed |
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| pubmed-article:16551349 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:16551349 | pubmed:volume | 7 | lld:pubmed |
| pubmed-article:16551349 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16551349 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16551349 | pubmed:pagination | 28 | lld:pubmed |
| pubmed-article:16551349 | pubmed:dateRevised | 2011-9-9 | lld:pubmed |
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| pubmed-article:16551349 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16551349 | pubmed:articleTitle | Paraoxonase gene polymorphisms and haplotype analysis in a stroke population. | lld:pubmed |
| pubmed-article:16551349 | pubmed:affiliation | Department of Medicine and Therapeutics, University of Aberdeen, UK. a.pasdar@abdn.ac.uk | lld:pubmed |
| pubmed-article:16551349 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16551349 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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