pubmed-article:16199828 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16199828 | lifeskim:mentions | umls-concept:C0439849 | lld:lifeskim |
pubmed-article:16199828 | lifeskim:mentions | umls-concept:C0036341 | lld:lifeskim |
pubmed-article:16199828 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
pubmed-article:16199828 | lifeskim:mentions | umls-concept:C1424906 | lld:lifeskim |
pubmed-article:16199828 | lifeskim:mentions | umls-concept:C0018591 | lld:lifeskim |
pubmed-article:16199828 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
pubmed-article:16199828 | lifeskim:mentions | umls-concept:C2348519 | lld:lifeskim |
pubmed-article:16199828 | pubmed:issue | 10 | lld:pubmed |
pubmed-article:16199828 | pubmed:dateCreated | 2005-10-3 | lld:pubmed |
pubmed-article:16199828 | pubmed:abstractText | The purpose of this study was to determine whether a haplotype in the dystrobrevin binding protein 1 (DTNBP1) gene previously associated with schizophrenia not only increases the susceptibility to psychotic illness but also to a more or less clinically specific form of psychotic illness. | lld:pubmed |
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pubmed-article:16199828 | pubmed:language | eng | lld:pubmed |
pubmed-article:16199828 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16199828 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:16199828 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16199828 | pubmed:month | Oct | lld:pubmed |
pubmed-article:16199828 | pubmed:issn | 0002-953X | lld:pubmed |
pubmed-article:16199828 | pubmed:author | pubmed-author:NealeMichael... | lld:pubmed |
pubmed-article:16199828 | pubmed:author | pubmed-author:KendlerKennet... | lld:pubmed |
pubmed-article:16199828 | pubmed:author | pubmed-author:van den... | lld:pubmed |
pubmed-article:16199828 | pubmed:author | pubmed-author:WalshDermotD | lld:pubmed |
pubmed-article:16199828 | pubmed:author | pubmed-author:O'NeillF... | lld:pubmed |
pubmed-article:16199828 | pubmed:author | pubmed-author:AggenSteven... | lld:pubmed |
pubmed-article:16199828 | pubmed:author | pubmed-author:FanousAyman... | lld:pubmed |
pubmed-article:16199828 | pubmed:author | pubmed-author:RileyBrien... | lld:pubmed |
pubmed-article:16199828 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16199828 | pubmed:volume | 162 | lld:pubmed |
pubmed-article:16199828 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16199828 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16199828 | pubmed:pagination | 1824-32 | lld:pubmed |
pubmed-article:16199828 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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pubmed-article:16199828 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:16199828 | pubmed:articleTitle | Relationship between a high-risk haplotype in the DTNBP1 (dysbindin) gene and clinical features of schizophrenia. | lld:pubmed |
pubmed-article:16199828 | pubmed:affiliation | Washington VA Medical Center-Georgetown University Medical Center Schizophrenia Research Program, 50 Irving St. NW, Washington, DC 20422, USA. ayman.fanous@med.va.gov | lld:pubmed |
pubmed-article:16199828 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16199828 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:16199828 | pubmed:publicationType | Multicenter Study | lld:pubmed |
pubmed-article:16199828 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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