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pubmed-article:16169484pubmed:abstractTextMalignant melanoma is a highly aggressive neoplastic disease whose incidence is increasing rapidly. In recent years, the use of interferon alpha (IFNalpha) has become the most established adjuvant immunotherapy for melanoma of advanced stage. IFNalpha is a potent inhibitor of melanoma cell proliferation, and the signal transducer and activator of transcription STAT1 is crucial for its antiproliferative action. Although advanced melanomas clinically resistant to IFNalpha are frequently characterized by inefficient STAT1 signaling, the mechanisms underlying advanced-stage interferon resistance are poorly understood.lld:pubmed
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pubmed-article:16169484pubmed:articleTitleSTAT5 contributes to interferon resistance of melanoma cells.lld:pubmed
pubmed-article:16169484pubmed:affiliationDepartment of Physiological Chemistry I, Biocenter, Theodor-Boveri Institute, University of Würzburg, Am Hubland, 97074 Würzburg, Germany. claudia.wellbrock@icr.ac.uklld:pubmed
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