| pubmed-article:14730619 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:14730619 | lifeskim:mentions | umls-concept:C1123023 | lld:lifeskim |
| pubmed-article:14730619 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
| pubmed-article:14730619 | lifeskim:mentions | umls-concept:C0040300 | lld:lifeskim |
| pubmed-article:14730619 | lifeskim:mentions | umls-concept:C0016059 | lld:lifeskim |
| pubmed-article:14730619 | lifeskim:mentions | umls-concept:C0003009 | lld:lifeskim |
| pubmed-article:14730619 | lifeskim:mentions | umls-concept:C0036421 | lld:lifeskim |
| pubmed-article:14730619 | lifeskim:mentions | umls-concept:C0529330 | lld:lifeskim |
| pubmed-article:14730619 | lifeskim:mentions | umls-concept:C1880177 | lld:lifeskim |
| pubmed-article:14730619 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:14730619 | pubmed:dateCreated | 2004-1-19 | lld:pubmed |
| pubmed-article:14730619 | pubmed:abstractText | Tissue fibrosis in systemic sclerosis (SSc) is attributed to excessive deposition of extracellular matrix components produced by fibroblasts in skin lesions. Angiotensin II (Ang II), a vasoconstrictive peptide, is reported to have profibrotic activity as a result of induction of the extracellular matrix. The aim of the present study was to examine the expression of Ang II and its type 1 (AT(1)) and type 2 (AT(2)) receptors in affected skin and dermal fibroblasts from patients with SSc and to study the role of Ang II in collagen production by SSc dermal fibroblasts. | lld:pubmed |
| pubmed-article:14730619 | pubmed:language | eng | lld:pubmed |
| pubmed-article:14730619 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:14730619 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:14730619 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:14730619 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:14730619 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:14730619 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:14730619 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:14730619 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:14730619 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:14730619 | pubmed:issn | 0004-3591 | lld:pubmed |
| pubmed-article:14730619 | pubmed:author | pubmed-author:KawaguchiYasu... | lld:pubmed |
| pubmed-article:14730619 | pubmed:author | pubmed-author:HarigaiMasayo... | lld:pubmed |
| pubmed-article:14730619 | pubmed:author | pubmed-author:HaraMasakoM | lld:pubmed |
| pubmed-article:14730619 | pubmed:author | pubmed-author:KamataniNaoyu... | lld:pubmed |
| pubmed-article:14730619 | pubmed:author | pubmed-author:SugiuraTomoko... | lld:pubmed |
| pubmed-article:14730619 | pubmed:author | pubmed-author:TakagiKaeK | lld:pubmed |
| pubmed-article:14730619 | pubmed:author | pubmed-author:FukasawaChika... | lld:pubmed |
| pubmed-article:14730619 | pubmed:author | pubmed-author:NishimagiEmiE | lld:pubmed |
| pubmed-article:14730619 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:14730619 | pubmed:volume | 50 | lld:pubmed |
| pubmed-article:14730619 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:14730619 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:14730619 | pubmed:pagination | 216-26 | lld:pubmed |
| pubmed-article:14730619 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:14730619 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:14730619 | pubmed:articleTitle | Angiotensin II in the lesional skin of systemic sclerosis patients contributes to tissue fibrosis via angiotensin II type 1 receptors. | lld:pubmed |
| pubmed-article:14730619 | pubmed:affiliation | Tokyo Women's Medical University School of Medicine, Tokyo, Japan. y-kawa@ior.twmu.ac.jp | lld:pubmed |
| pubmed-article:14730619 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:14730619 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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