| pubmed-article:12887886 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12887886 | lifeskim:mentions | umls-concept:C0008633 | lld:lifeskim |
| pubmed-article:12887886 | lifeskim:mentions | umls-concept:C0040649 | lld:lifeskim |
| pubmed-article:12887886 | lifeskim:mentions | umls-concept:C1514562 | lld:lifeskim |
| pubmed-article:12887886 | lifeskim:mentions | umls-concept:C1883221 | lld:lifeskim |
| pubmed-article:12887886 | lifeskim:mentions | umls-concept:C1883204 | lld:lifeskim |
| pubmed-article:12887886 | lifeskim:mentions | umls-concept:C1880389 | lld:lifeskim |
| pubmed-article:12887886 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:12887886 | pubmed:dateCreated | 2003-7-30 | lld:pubmed |
| pubmed-article:12887886 | pubmed:abstractText | The histone variant macroH2A plays a role in mammalian chromosome X inactivation. Recent data suggest its unusual C-terminal macro domain may be an enzyme that regulates the ADP-ribosylation of chromatin. MacroH2A could thus help correct gene dosage between males and females using a novel epigenetic mark. | lld:pubmed |
| pubmed-article:12887886 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12887886 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12887886 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:12887886 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12887886 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:12887886 | pubmed:issn | 1097-2765 | lld:pubmed |
| pubmed-article:12887886 | pubmed:author | pubmed-author:LadurnerAndre... | lld:pubmed |
| pubmed-article:12887886 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12887886 | pubmed:volume | 12 | lld:pubmed |
| pubmed-article:12887886 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12887886 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12887886 | pubmed:pagination | 1-3 | lld:pubmed |
| pubmed-article:12887886 | pubmed:dateRevised | 2005-11-17 | lld:pubmed |
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| pubmed-article:12887886 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:12887886 | pubmed:articleTitle | Inactivating chromosomes: a macro domain that minimizes transcription. | lld:pubmed |
| pubmed-article:12887886 | pubmed:affiliation | Gene Expression Programme and Structural and Computational Biology Programme, European Molecular Biology Laboratory, Meyerhofstrasse 1, 69117 Heidelberg, Germany. | lld:pubmed |
| pubmed-article:12887886 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12887886 | pubmed:publicationType | Review | lld:pubmed |
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