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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
6
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pubmed:dateCreated |
2002-11-12
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pubmed:abstractText |
It is known that cyclosporine A (CsA) treatment induces high bone-turnover osteopenia and hypercalciuria. It has been proposed that down-regulation of renal calbindin-D28k by CsA results in renal calcium wasting. We investigated the role of the kidney and bone in CsA-induced hypercalciuria in calbindin-D28k knockout (KO) and wild-type (WT) mice.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
0085-2538
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
62
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
2055-61
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:12427129-Animals,
pubmed-meshheading:12427129-Anti-Inflammatory Agents,
pubmed-meshheading:12427129-Bone Diseases, Metabolic,
pubmed-meshheading:12427129-Calcium,
pubmed-meshheading:12427129-Calcium Channels,
pubmed-meshheading:12427129-Calcium-Binding Protein, Vitamin D-Dependent,
pubmed-meshheading:12427129-Cyclosporine,
pubmed-meshheading:12427129-Diphosphonates,
pubmed-meshheading:12427129-Gene Expression,
pubmed-meshheading:12427129-Immunosuppressive Agents,
pubmed-meshheading:12427129-Mice,
pubmed-meshheading:12427129-Mice, Inbred C57BL,
pubmed-meshheading:12427129-Mice, Knockout
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pubmed:year |
2002
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pubmed:articleTitle |
Cyclosporine A-induced hypercalciuria in calbindin-D28k knockout and wild-type mice.
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pubmed:affiliation |
Department of Medicine, University of Arizona Health Sciences Centers, Tucson, Arizona 85724, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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