pubmed-article:12199067 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:12199067 | lifeskim:mentions | umls-concept:C0030567 | lld:lifeskim |
pubmed-article:12199067 | lifeskim:mentions | umls-concept:C1882598 | lld:lifeskim |
pubmed-article:12199067 | lifeskim:mentions | umls-concept:C0028027 | lld:lifeskim |
pubmed-article:12199067 | lifeskim:mentions | umls-concept:C1947976 | lld:lifeskim |
pubmed-article:12199067 | lifeskim:mentions | umls-concept:C1154437 | lld:lifeskim |
pubmed-article:12199067 | lifeskim:mentions | umls-concept:C1517004 | lld:lifeskim |
pubmed-article:12199067 | lifeskim:mentions | umls-concept:C0068728 | lld:lifeskim |
pubmed-article:12199067 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:12199067 | pubmed:dateCreated | 2002-8-29 | lld:pubmed |
pubmed-article:12199067 | pubmed:abstractText | It was established, that serotonin and dopamine content and dopamine uptake by brain nerve endings under experimental parkinsonism are decreased. Nicotinamide nicotinoyl-GABA administration leads to normalization these parameters. It was shown that NAm was more effective on serotonin content while nicotinoyl-GABA on dopamine one. Dopamine uptake was impaired at experimental parkinsonism and partially was normalized by incubation with NAD (10(-6) M). Thus, NAm, nicotinoyl-GABA and NAD are involved in the regulation of brain neurotransmission under experimental parkinsonism and can be useful in treatment of Parkinson's disease. | lld:pubmed |
pubmed-article:12199067 | pubmed:language | ukr | lld:pubmed |
pubmed-article:12199067 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12199067 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:12199067 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12199067 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12199067 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12199067 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:12199067 | pubmed:issn | 0201-8470 | lld:pubmed |
pubmed-article:12199067 | pubmed:author | pubmed-author:FomenkoH IHI | lld:pubmed |
pubmed-article:12199067 | pubmed:author | pubmed-author:Kuchmerovs'ka... | lld:pubmed |
pubmed-article:12199067 | pubmed:author | pubmed-author:DonchenkoH... | lld:pubmed |
pubmed-article:12199067 | pubmed:author | pubmed-author:Chichkovs'kaH... | lld:pubmed |
pubmed-article:12199067 | pubmed:author | pubmed-author:PakirbaievaL... | lld:pubmed |
pubmed-article:12199067 | pubmed:author | pubmed-author:KlymenkoA NAN | lld:pubmed |
pubmed-article:12199067 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:12199067 | pubmed:volume | 73 | lld:pubmed |
pubmed-article:12199067 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:12199067 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:12199067 | pubmed:pagination | 108-12 | lld:pubmed |
pubmed-article:12199067 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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pubmed-article:12199067 | pubmed:articleTitle | [Correction by nicotinamide and nicotinoyl-GABA of dopamine metabolism in rat brain in experimental Parkinson's disease]. | lld:pubmed |
pubmed-article:12199067 | pubmed:affiliation | Palladin Institute of Biochemistry, NAS of Ukraine, Kyiv. kuch@biochem.kiev.ua | lld:pubmed |
pubmed-article:12199067 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:12199067 | pubmed:publicationType | English Abstract | lld:pubmed |