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| pubmed-article:12195127 | lifeskim:mentions | umls-concept:C0034705 | lld:lifeskim |
| pubmed-article:12195127 | lifeskim:mentions | umls-concept:C0028128 | lld:lifeskim |
| pubmed-article:12195127 | lifeskim:mentions | umls-concept:C0227651 | lld:lifeskim |
| pubmed-article:12195127 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:12195127 | lifeskim:mentions | umls-concept:C1817184 | lld:lifeskim |
| pubmed-article:12195127 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:12195127 | pubmed:dateCreated | 2002-8-26 | lld:pubmed |
| pubmed-article:12195127 | pubmed:abstractText | Cellular responses to agonists of G protein-coupled receptors are usually rapidly attenuated - a process known as 'receptor desensitization'. The mechanisms that attenuate signalling are important both physiologically and therapeutically. | lld:pubmed |
| pubmed-article:12195127 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12195127 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:12195127 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:12195127 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12195127 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:12195127 | pubmed:issn | 0263-6352 | lld:pubmed |
| pubmed-article:12195127 | pubmed:author | pubmed-author:LiuRuishengR | lld:pubmed |
| pubmed-article:12195127 | pubmed:author | pubmed-author:PerssonA... | lld:pubmed |
| pubmed-article:12195127 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12195127 | pubmed:volume | 20 | lld:pubmed |
| pubmed-article:12195127 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12195127 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12195127 | pubmed:pagination | 1835-42 | lld:pubmed |
| pubmed-article:12195127 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:12195127 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12195127 | pubmed:articleTitle | Effects of nitric oxide on P2Y receptor resensitization in spontaneously hypertensive rat mesangial cells. | lld:pubmed |
| pubmed-article:12195127 | pubmed:affiliation | Department of Medical Cell Biology, Uppsala University, Sweden. | lld:pubmed |
| pubmed-article:12195127 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12195127 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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