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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2002-7-3
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pubmed:abstractText |
Hyperhomocysteinemia has been established as an important risk factor for cardiovascular diseases. The aim of the present study was to investigate whether hyperhomocysteinemia and/or homozygosity for the methylenetetrahydrofolate reductase (MTHFR) C677T mutation are associated with an increased risk for retinal artery occlusion (RAO).
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
0002-9394
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
134
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
57-61
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pubmed:dateRevised |
2005-11-17
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pubmed:meshHeading |
pubmed-meshheading:12095808-Adult,
pubmed-meshheading:12095808-Aged,
pubmed-meshheading:12095808-Aged, 80 and over,
pubmed-meshheading:12095808-Case-Control Studies,
pubmed-meshheading:12095808-Chromatography, High Pressure Liquid,
pubmed-meshheading:12095808-DNA,
pubmed-meshheading:12095808-Female,
pubmed-meshheading:12095808-Genotype,
pubmed-meshheading:12095808-Homocysteine,
pubmed-meshheading:12095808-Humans,
pubmed-meshheading:12095808-Hyperhomocysteinemia,
pubmed-meshheading:12095808-Male,
pubmed-meshheading:12095808-Methylenetetrahydrofolate Reductase (NADPH2),
pubmed-meshheading:12095808-Middle Aged,
pubmed-meshheading:12095808-Oxidoreductases Acting on CH-NH Group Donors,
pubmed-meshheading:12095808-Point Mutation,
pubmed-meshheading:12095808-Polymerase Chain Reaction,
pubmed-meshheading:12095808-Retinal Artery Occlusion,
pubmed-meshheading:12095808-Retrospective Studies,
pubmed-meshheading:12095808-Risk Factors
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pubmed:year |
2002
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pubmed:articleTitle |
The role of hyperhomocysteinemia and methylenetetrahydrofolate reductase (MTHFR) C677T mutation in patients with retinal artery occlusion.
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pubmed:affiliation |
Department of Ophthalmology, Karl-Franzens University, Graz, Austria. martin.weger@kfunigraz.ac.at
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pubmed:publicationType |
Journal Article
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