11833998

Source:http://linkedlifedata.com/resource/pubmed/id/11833998

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0007202, umls-concept:C0010055, umls-concept:C0543467, umls-concept:C2246421
pubmed:issue	1
pubmed:dateCreated	2002-2-8
pubmed:abstractText	Granulocyte apoptosis is a key control process in the clearance of neutrophils from inflammatory sites, and its rate is modulated both in vitro and in vivo by a number of inflammatory mediators. In this study, we investigated the influence of cardiopulmonary bypass (CPB) on neutrophil apoptosis.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/15030100R
pubmed:citationSubset	AIM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD95, http://linkedlifedata.com/resource/pubmed/chemical/Caspases, http://linkedlifedata.com/resource/pubmed/chemical/Cytokines, http://linkedlifedata.com/resource/pubmed/chemical/FASLG protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Fas Ligand Protein, http://linkedlifedata.com/resource/pubmed/chemical/Inflammation Mediators, http://linkedlifedata.com/resource/pubmed/chemical/Interleukin-6, http://linkedlifedata.com/resource/pubmed/chemical/Interleukin-8, http://linkedlifedata.com/resource/pubmed/chemical/Membrane Glycoproteins, http://linkedlifedata.com/resource/pubmed/chemical/Tumor Necrosis Factor-alpha
pubmed:status	MEDLINE
pubmed:month	Jan
pubmed:issn	0003-4975
pubmed:author	pubmed-author:ChelloMassimoM, pubmed-author:CirilloFrancescoF, pubmed-author:CovinoElvioE, pubmed-author:CudaGiovanniG, pubmed-author:MastrorobertoPasqualeP, pubmed-author:PerticoneFrancescoF, pubmed-author:QuirinoAngelaA
pubmed:issnType	Print
pubmed:volume	73
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	123-9; discussion 129-30
pubmed:dateRevised	2006-11-15
pubmed:meshHeading	pubmed-meshheading:11833998-Aged, pubmed-meshheading:11833998-Antigens, CD95, pubmed-meshheading:11833998-Apoptosis, pubmed-meshheading:11833998-Cardiopulmonary Bypass, pubmed-meshheading:11833998-Caspases, pubmed-meshheading:11833998-Coronary Artery Bypass, pubmed-meshheading:11833998-Cytokines, pubmed-meshheading:11833998-Fas Ligand Protein, pubmed-meshheading:11833998-Female, pubmed-meshheading:11833998-Humans, pubmed-meshheading:11833998-Inflammation Mediators, pubmed-meshheading:11833998-Interleukin-6, pubmed-meshheading:11833998-Interleukin-8, pubmed-meshheading:11833998-Male, pubmed-meshheading:11833998-Membrane Glycoproteins, pubmed-meshheading:11833998-Middle Aged, pubmed-meshheading:11833998-Neutrophil Activation, pubmed-meshheading:11833998-Neutrophils, pubmed-meshheading:11833998-Postoperative Period, pubmed-meshheading:11833998-Tumor Necrosis Factor-alpha
pubmed:year	2002
pubmed:articleTitle	Inhibition of neutrophil apoptosis after coronary bypass operation with cardiopulmonary bypass.
pubmed:affiliation	Department of Clinical and Experimental Medicine, Unit of Cardiac Surgery, Medical School of Catanzaro, Italy. chello@unicz.it
pubmed:publicationType	Journal Article