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| pubmed-article:10892870 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
| pubmed-article:10892870 | lifeskim:mentions | umls-concept:C0042666 | lld:lifeskim |
| pubmed-article:10892870 | lifeskim:mentions | umls-concept:C0023317 | lld:lifeskim |
| pubmed-article:10892870 | lifeskim:mentions | umls-concept:C0271160 | lld:lifeskim |
| pubmed-article:10892870 | lifeskim:mentions | umls-concept:C1330957 | lld:lifeskim |
| pubmed-article:10892870 | lifeskim:mentions | umls-concept:C0033684 | lld:lifeskim |
| pubmed-article:10892870 | lifeskim:mentions | umls-concept:C0596311 | lld:lifeskim |
| pubmed-article:10892870 | lifeskim:mentions | umls-concept:C0596235 | lld:lifeskim |
| pubmed-article:10892870 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:10892870 | lifeskim:mentions | umls-concept:C1517945 | lld:lifeskim |
| pubmed-article:10892870 | lifeskim:mentions | umls-concept:C0449584 | lld:lifeskim |
| pubmed-article:10892870 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:10892870 | pubmed:dateCreated | 2000-7-31 | lld:pubmed |
| pubmed-article:10892870 | pubmed:abstractText | Cortical cataract in humans is associated with Ca2+ overload and protein loss, and although animal models of cataract have implicated Ca2+-activated proteases in this process, it remains to be determined whether the human lens responds in this manner to conditions of Ca2+ overload. The purpose of these experiments was to investigate Ca2+-induced opacification and proteolysis in the organ-cultured human lens. | lld:pubmed |
| pubmed-article:10892870 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10892870 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10892870 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10892870 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10892870 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10892870 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:10892870 | pubmed:issn | 0146-0404 | lld:pubmed |
| pubmed-article:10892870 | pubmed:author | pubmed-author:DuncanGG | lld:pubmed |
| pubmed-article:10892870 | pubmed:author | pubmed-author:SandersonJJ | lld:pubmed |
| pubmed-article:10892870 | pubmed:author | pubmed-author:MarcantonioJ... | lld:pubmed |
| pubmed-article:10892870 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10892870 | pubmed:volume | 41 | lld:pubmed |
| pubmed-article:10892870 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10892870 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10892870 | pubmed:pagination | 2255-61 | lld:pubmed |
| pubmed-article:10892870 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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| pubmed-article:10892870 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:10892870 | pubmed:articleTitle | A human lens model of cortical cataract: Ca2+-induced protein loss, vimentin cleavage and opacification. | lld:pubmed |
| pubmed-article:10892870 | pubmed:affiliation | School of Biological Sciences, University of East Anglia, Norwich, United Kingdom. j.sanderson@uea.ac.uk | lld:pubmed |
| pubmed-article:10892870 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10892870 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:10892870 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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