rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2000-3-13
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pubmed:abstractText |
The causes of volatile anesthetic-induced cerebral vasodilation include direct effects on smooth muscle and indirect effects via changes in metabolic rate and release of mediators from vascular endothelium and brain parenchyma. The role of nitric oxide and the relative importance of neuronal and endothelial nitric oxide synthase (nNOS and eNOS, respectively) are unclear.
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pubmed:grant |
|
pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
|
pubmed:issn |
0003-3022
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pubmed:author |
|
pubmed:issnType |
Print
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pubmed:volume |
92
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
|
pubmed:pagination |
125-32
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:10638908-Analysis of Variance,
pubmed-meshheading:10638908-Anesthetics, Inhalation,
pubmed-meshheading:10638908-Animals,
pubmed-meshheading:10638908-Brain,
pubmed-meshheading:10638908-Cerebrovascular Circulation,
pubmed-meshheading:10638908-Drug Interactions,
pubmed-meshheading:10638908-Enzyme Inhibitors,
pubmed-meshheading:10638908-Halothane,
pubmed-meshheading:10638908-Indazoles,
pubmed-meshheading:10638908-Male,
pubmed-meshheading:10638908-Microcirculation,
pubmed-meshheading:10638908-Muscle, Smooth, Vascular,
pubmed-meshheading:10638908-NG-Nitroarginine Methyl Ester,
pubmed-meshheading:10638908-Nitric Oxide,
pubmed-meshheading:10638908-Nitric Oxide Synthase,
pubmed-meshheading:10638908-Rats,
pubmed-meshheading:10638908-Rats, Sprague-Dawley,
pubmed-meshheading:10638908-Vasodilation
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pubmed:year |
2000
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pubmed:articleTitle |
Neuronal nitric oxide synthase mediates halothane-induced cerebral microvascular dilation.
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pubmed:affiliation |
Department of Anesthesiology, Medical College of Wisconsin and the Children's Hospital of Wisconsin, Milwaukee 53226, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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