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| pubmed-article:10441517 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:10441517 | pubmed:dateCreated | 1999-9-9 | lld:pubmed |
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| pubmed-article:10441517 | pubmed:abstractText | Inactivation of the p53 tumor suppressor protein has been observed in a large number of human cancers. Overexpression of p53 induces either growth arrest or programmed cell death (apoptosis). The growth arrest function of p53 is mediated by induction of p21 (WAF1/CIP1), but the mechanisms underlying p53-dependent apoptosis are still largely unknown. To investigate these mechanisms, we have identified six differentially expressed transcripts in a human colon cancer cell line undergoing p53-dependent apoptosis. One of the p53-responsive genes showed significant homology to Drosophila peroxidasin, an extracellular matrix-associated peroxidase, and is likely to be its human homologue. Our results suggest a possible connection between p53-dependent apoptosis and the production of reactive oxygen species. | lld:pubmed |
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| pubmed-article:10441517 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:10441517 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10441517 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10441517 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:10441517 | pubmed:issn | 0006-291X | lld:pubmed |
| pubmed-article:10441517 | pubmed:author | pubmed-author:HorikoshiNN | lld:pubmed |
| pubmed-article:10441517 | pubmed:author | pubmed-author:ShenkTT | lld:pubmed |
| pubmed-article:10441517 | pubmed:author | pubmed-author:ConeLL | lld:pubmed |
| pubmed-article:10441517 | pubmed:author | pubmed-author:KleyNN | lld:pubmed |
| pubmed-article:10441517 | pubmed:copyrightInfo | Copyright 1999 Academic Press. | lld:pubmed |
| pubmed-article:10441517 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10441517 | pubmed:day | 11 | lld:pubmed |
| pubmed-article:10441517 | pubmed:volume | 261 | lld:pubmed |
| pubmed-article:10441517 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10441517 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10441517 | pubmed:pagination | 864-9 | lld:pubmed |
| pubmed-article:10441517 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:10441517 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10441517 | pubmed:articleTitle | Isolation of differentially expressed cDNAs from p53-dependent apoptotic cells: activation of the human homologue of the Drosophila peroxidasin gene. | lld:pubmed |
| pubmed-article:10441517 | pubmed:affiliation | Department of Medicine, Beth Israel Deaconess Medical Center, Boston, Massachusetts, 02115, USA. nhorikos@caregroup.harvard.edu | lld:pubmed |
| pubmed-article:10441517 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10441517 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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