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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
2
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pubmed:dateCreated |
1999-8-30
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pubmed:abstractText |
Angiotensin type 1 (AT1) receptor-deficient mice (Agtr1-/-), which selectively lack both AT1A and AT1B receptor genes, are characterized by marked intrarenal vascular thickening. In the present study, we explored the possible involvement of the kinin-kallikrein system in the development of this renal vascular hypertrophy.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Aprotinin,
http://linkedlifedata.com/resource/pubmed/chemical/Kallikreins,
http://linkedlifedata.com/resource/pubmed/chemical/Peptidyl-Dipeptidase A,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger,
http://linkedlifedata.com/resource/pubmed/chemical/Receptor, Angiotensin, Type 1,
http://linkedlifedata.com/resource/pubmed/chemical/Receptor, Angiotensin, Type 2,
http://linkedlifedata.com/resource/pubmed/chemical/Receptor, Bradykinin B2,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Angiotensin,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Bradykinin,
http://linkedlifedata.com/resource/pubmed/chemical/Serine Proteinase Inhibitors
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pubmed:status |
MEDLINE
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pubmed:month |
Aug
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pubmed:issn |
0085-2538
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
56
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
509-16
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:10432390-Animals,
pubmed-meshheading:10432390-Aprotinin,
pubmed-meshheading:10432390-Arterioles,
pubmed-meshheading:10432390-Female,
pubmed-meshheading:10432390-Gene Expression Regulation, Developmental,
pubmed-meshheading:10432390-Hypertrophy,
pubmed-meshheading:10432390-Immunohistochemistry,
pubmed-meshheading:10432390-Kallikrein-Kinin System,
pubmed-meshheading:10432390-Kallikreins,
pubmed-meshheading:10432390-Kidney,
pubmed-meshheading:10432390-Male,
pubmed-meshheading:10432390-Mice,
pubmed-meshheading:10432390-Mice, Inbred C57BL,
pubmed-meshheading:10432390-Mice, Knockout,
pubmed-meshheading:10432390-Peptidyl-Dipeptidase A,
pubmed-meshheading:10432390-RNA, Messenger,
pubmed-meshheading:10432390-Receptor, Angiotensin, Type 1,
pubmed-meshheading:10432390-Receptor, Angiotensin, Type 2,
pubmed-meshheading:10432390-Receptor, Bradykinin B2,
pubmed-meshheading:10432390-Receptors, Angiotensin,
pubmed-meshheading:10432390-Receptors, Bradykinin,
pubmed-meshheading:10432390-Renal Artery,
pubmed-meshheading:10432390-Renal Circulation,
pubmed-meshheading:10432390-Renin-Angiotensin System,
pubmed-meshheading:10432390-Serine Proteinase Inhibitors
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pubmed:year |
1999
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pubmed:articleTitle |
Potent antihypertrophic effect of the bradykinin B2 receptor system on the renal vasculature.
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pubmed:affiliation |
Department of Pediatrics, Vanderbilt University Medical Center, Nashville, Tennessee 37232-2584, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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